16 Antioxidant Herbs for Alzheimer’s on Your Spice Rack

Antioxidant Herbs for Alzheimer's

Researchers have not yet been able to say definitively what the cause of Alzheimer’s disease is. But, among the candidate causes discussed is oxidation in the body – also called “oxidative stress.”

I want to survey the antioxidant potential of sixteen (16) herbs that you might have on your spice rack at home.

The Relevance of Oxidation

Oxidative stress dovetails with Alzheimer’s disease in several respects.

Firstly, patients who already have Alzheimer’s disease have problems with accelerated oxidation. They have more oxidative stress in their brains.

Secondly, many readers may also be aware that one of the primary characteristics of Alzheimer’s disease is that there is an abnormal accumulation of beta-amyloid and tau protein deposits, which aggregate between – and inside of – nerve cells, gumming up the neural “works.” Well, this abnormal deposition of protein has been speculated either to be the result of an increase in “free radicals” or a decrease in antioxidant defenses inside of some people’s bodies. In other words, it’s the result of oxidation.

But that raises the question: Are antioxidant therapies for Alzheimer’s disease viable?

In attempting to answer this question, we should be mindful of the fact that herbs can be excellent sources for antioxidant. And, as I mentioned at the outset, many of these versatile plants can be found in your kitchen.

In this article, I’ll list sixteen of the most common. (In a follow-up post, I’ll provide a further sixteen that, while less common, are still available.)

Caveats

Obviously, I’m not guaranteeing that you will have all – or even any of – these on your spice rack. Various starter sets will likely include some elements that I don’t write about. Contrariwise, your set may fail to have some component that I do discuss.

Additionally, these profiles are not exhaustive. Moreover, not every example of these herbs will have exactly the same chemical constituents or in the same amounts. It often depends on how the plants are grown, what weather conditions were like, how they were harvested, how the extract was harvested, how was stored, etc. Numerous peer-reviewed scientific journals contain more detailed information on herbs, in general, and on their antioxidant constituents, specifically.

16 Common Antioxidant Herbs You Have on Your Kitchen Spice Rack

Spice #1: Basil[1] (Ocimum basilicum)

Like most of the herbs surveyed here, basil is a good source of vitamins – many of which actually are antioxidants, themselves. But what I’m going to be concerned with is some of the other antioxidant chemicals that are found in these plants.

Basil’s Antioxidant Profile

I have come up with these little graphics with a list of antioxidants. I’m calling these the “antioxidant profile.”

In some cases, an herb’s antioxidant activity can’t be explained solely by the presence of any single compound on the list. In other words, following this line of thinking, it’s not just eugenol, but it’s eugenol in conjunction with all of the other chemicals contained in basil.

(Note: eugenol is more abundantly present in oil of clove. See Part 2.)

This propensity of antioxidants to complement and mutually amplify one another is referred to as “synergism.”

In addition to basil’s significant antioxidants, the herb has also been shown to increase both “memory retention” and “memory retrieval” in experiments on mice.[2]

Spice #2: Bay Laurel Leaves (Laurus nobilis)

Bay Laurel’s Antioxidant Profile

You can see that my list for bay leaves is a little bit less expansive than it was for basil. sale being the primary one.

Nevertheless, bay laurel essential oil is a rich source of natural antioxidants. In fact, the anti-inflammatory and antioxidant potential of “laurel extracts is very significant,” particularly in relation to pathologies such as “Parkinson’s disease, Alzheimer’s disease, and atherosclerosis.”[3]

Spice #3: Black Pepper (Piper nigrum)

Black Pepper’s Antioxidant Profile

This one is really interesting to me. I’m fascinated that this is on the list because even people who don’t have a “collection” of herbs and spices probably have a salt and pepper shaker!

So, it’s amazing that pepper – regular pepper – may have many health-promoting qualities. And it may be highly relevant to Alzheimer’s prevention and treatment.[4]

For example, it’s full of antioxidants.

Piper nigrum’s signature component is something termed “piperine.” You don’t have to go further than the title of one Food-and-Chemical-Toxicology article to get a sense of why this is constituent is so exciting. “Piperine, the main alkaloid of …black pepper, protects against neurodegeneration and cognitive impairment in animal model[s] of cognitive deficit[s] like …Alzheimer’s disease.”[5]

It might be worthwhile to develop a taste for black pepper – if you don’t have one already! Sprinkle a little on your food next dinnertime.

Not only is a good antioxidant, in general, but it actually helps reduce high-fat-intake-induced oxidative stress, specifically.[6] Obesity, and consumption of high-fat diets are both known to be risk factors – that is, increase risk – for Alzheimer’s.

I get into this further in Part 1 of my YouTube-video series on sugar and dementia. (Watch Part 1, HERE.)

But, since black pepper is also purported to have acetylcholinesterase activity,[7] Piper nigrum is also an excellent and promising candidate for multi-target Alzheimer’s-disease therapies.

(And… if you want to get more into acetylcholinesterase inhibitors, I have separate YouTube videos on that subject. For a rudimentary explanation of how acetylcholinesterase inhibitors are supposed to work, see HERE. On the perhaps far-fetched and – as far as I know – merely theoretical possibility that a natural “acetylcholinesterase deficiency” might be beneficial in avoiding Alzheimer’s, see HERE. Finally, on the six, Food-and-Drug-Administration-approved Alzheimer’s drugs – five of which have acetylcholinesterase-inhibiting functions – see HERE.)

It’s worth observing that it’s not just black pepper that is reported to deliver positive health effects. The Piper genus has some 2,000 species of plants.[8] It is plausible to think that at least some of these will have effects similar to those of black pepper just rehearsed.

Spice #4: Cayenne / Paprika (Capsicum annuum)

Cayenne / Paprika’s Antioxidant Profile

Speaking of varieties of pepper, cayenne is another one commonly locatable on kitchen spice racks.

Rich in antioxidants, Cayenne’s main constituents include ascorbic acid, which is (of course) the more “sciency” name for Vitamin C. (By the way, for more on Vitamin C, glance see further down in this article.)

Vitamin C isn’t this pepper’s only arrow in its nutritional quiver. Cayenne also contains a healthy quantity of calciferol, that is, Vitamin D.[9]

Cayenne is known, in part, for is its antidiabetic effect.[10] Of course, diabetes may increase a person’s risk of Alzheimer’s.[11]

I’ll add that some people might have a cannister labeled paprika in their herbal starter packs. Now, I am not a chef, so this is not a culinary observation, but… cayenne and paprika are very similar. In fact, they’re similar enough that, from our standpoint, they can be considered readily interchangeable.[12]

So, for example, paprika is an antioxidant. It contains a variety of compounds, including Vitamin A – or “retinol” – and some other carotenoids, similarly to cayenne. It’s rich in antioxidants.[13]

You can see some validation of this in the article with the somewhat forbidding title, “Binding Antioxidant and Antiproliferative Properties of Sweet Paprika.”[14]

I’ll presently toss in a little caveat. Weather conditions as well as the various, possible ripening stages of individual peppers are going to impact the chemical constituents of these herbs. In the article cited in my footnote, you can see this point in reference to paprika.[15] However, as hinted at in my “disclaimers,” similar statements considerations apply to any of the herbs surveyed.

Spice #5: Cinnamon (Cinnamomum verum)[16]

Cinnamon’s Antioxidant Profile

Cinnamon is loaded with antioxidants, according to the commonly cited website Healthline.[17]

In some ways, cinnamon’s antioxidant effects are similar to those of black cardamom (Amomum subulatum),[18] which I’ll get into in Part 2. (Again, to see this presentation in a YouTube-video format, see HERE.)

Spice #6: Coriander Seeds [19] (Coriandrum sativum)

Coriander Seed’s Antioxidant Profile

Coriander seeds have potential as natural antioxidants.[20] In fact, in addition to coriander’s ability to counteract oxidative stress, the seeds also show antihyperglycemic activity.[21] This makes Coriandrum sativum yet another potential herbal prophylaxis against diabetes. (And this, in turn, may help to reduce a person’s risk of dementia.[22])

Beyond these properties, “Coriandrum sativum seed extract” appears directly relevant to Alzheimer’s and other forms of dementia (for example, Multi-Infarct Dementia, also called “Vascular Dementia”) in virtue of its observed “ameliorative effects on memory impairment in… mice.”[23]

To put it slightly differently, it can possibly “[repair] memory deficits”[24] – which is cause for excitement!

Spice #7: Cumin (Cuminum cyminum)

Cumin’s Antioxidant Profile

“…[C]umin is a potent antioxidant.”[25]

Its constituents include beta-carotene, cineole, and limonene.

According to an article in the peer-reviewed journal Pharmaceutical Biology, cumin also has antistress potential. But, interestingly, especially for the focus of this website, the authors enthuse that cumin also has “memory-enhancing activity.”[26]

They wrote: “This study provides scientific support for the antistress, antioxidant, and memory-enhancing activities of cumin extract and substantiates that its traditional use as a culinary spice in foods is beneficial and scientific in combating stress and related disorders.”[27]

It happens to be an excellent stand-in for coriander. So, if you don’t have coriander seeds, but you do have cumin, you’re in luck. Not only can you expect that cumin is substitutable from a culinary standpoint, but – as alluded to – it also can be used as a stand-in from the standpoint of antioxidant profiles.

Spice #8: Garlic (Allium sativum)

Garlic’s Antioxidant Profile

Speaking of those antioxidant profiles, garlic has a unique one.

Among its noteworthy chemical parts, garlic’s “signature” antioxidant is something called allicin.

Firstly, allicin is being investigated for its possible dual ability to “reduce neuronal death and ameliorate …spatial memory impairment in Alzheimer’s disease models.”[28]

Additionally, allicin also holds promise as an acetylcholinesterase inhibitor[29] – which is the main therapeutic action of the majority of Alzheimer’s drugs – including the preeminent donepezil (sold as Aricept) – currently prescribed.

We already touched on this a bit with black pepper (see above). And we’ll get into it again when we talk about sage (see below), which is also reported to have exceptional acetylcholinesterase-inhibiting activity.[30]

(For my YouTube video on how acetylcholinesterase-inhibiting pharmaceuticals work, click HERE. For my presentation on the six FDA-approved for Alzheimer’s, see HERE.)

In addition to its own antioxidant constituents, Aged Garlic Extract (AGE) has “the ability …to increase the levels of some antioxidant enzymes.”[31]

Among other things, garlic also has a storied history as an antibacterial agent. For more on the herb that, before Alexander Fleming arrived on the scene, was referred to “Russian Penicillin,” see, for example, the article “Extracts From the History and Medical Properties of Garlic.”[32]

Spice #9: Ginger (Zingiber officinale)

Ginger’s Antioxidant Profile

“Ginger is loaded with antioxidants…”.[33]

Recalling from our discussions of cayenne and cinnamon that diabetes is an Alzheimer’s risk factor, it’s also notable that “[g]inger has been shown to possess anti-diabetic activity in a variety of studies.”[34]

It also turns out that ginger has health-giving properties that are particularly relevant to females of the species. According to the peer-reviewed journal Evidence-Based Complementary and Alternative Medicine: “[G]inger is a potential cognitive enhancer for middle-aged women.”[35]

Finally, for Alzheimer’s, in particular, “Z. officinale may be a promising source of AChE [i.e., acetylcholinesterase – editor] inhibitors for Alzheimer’s disease.”[36] Indeed, some researchers think ginger may spark new insights into multi-targeted pharmaceutical approaches.[37]

Spice #10: Mustard (Brassica nigra)

Mustard’s Antioxidant Profile

It’s easy to dismiss mustard as a mere condiment. But, in fact, “Brassica nigra” is a remarkable “natural food source for antioxidants.”[38]

Healthline elaborates, stating: “Mustard contains antioxidants and other beneficial plant compounds thought to help protect your body against damage and disease. [i]t’s a great source of glucosinolates, a group of sulfur-containing compounds found in all cruciferous vegetables, including broccoli, cabbage, Brussels sprouts, and mustard.”[39]

Its antioxidant profile includes carotenes and kaempferol.

In the article titled “Kaempferol Attenuates Cognitive Deficit Via Regulating Oxidative Stress and Neuroinflammation…,”[40] appearing in the scientific journal Neural Regeneration Research, investigators report “that [kaempferol] may be a potential neuroprotective agent against cognitive deficit in [Alzheimer’s Disease].

As one of the most popular and widely used spices in the world, mustard is readily incorporated into a wide variety of dishes, recipes, and other culinary applications.

That said, however, bear in mind that various mustard-based preparations may include other ingredients besides the bare mustard seeds. Though, admittedly, some of these – for example, turmeric (see my YouTube videos HERE and HERE) and vinegar – may have salubrious properties of their own.

Spice #11: Nutmeg (Myristica fragrans)

Nutmeg’s Antioxidant Profile

Nutmeg is an interesting entrant to this list. First, let’s look at its “pluses.”

Predictably, the stuff “[c]ontains powerful antioxidants.”[41] As Healthline puts it: “Nutmeg contains an abundance of antioxidants, including plant pigments like cyanidins, essential oils, such as phenylpropanoids and terpenes, and phenolic compounds, including protocatechuic, ferulic, and caffeic acids.”[42]

Among its various “catechins,” and other assorted constituents – which together give it a free-radical-scavenging efficiency – is a unique chemical called “myristic acid.”

In one experiment, which involved feeding mice a “ketogenic diet …rich in myristic acid,” the studied diet “…significantly reduced total brain Aβ levels by approximately 25%.”[43]

Of course, the phrase “brain Aβ levels” refers to the pathological accumulation of junk, known as “beta-amyloid protein,” in the brains of Alzheimer’s suffers. These protein deposits are believed by some researchers to be the at-bottom cause of the dread disease. But, the jury is still out.

(For my own discussions of candidate Alzheimer’s-Disease causes, I invite you to see my WRITTEN ARTICLE elsewhere on this website or, if you’d prefer, view one of my early YOUTUBE-VIDEO efforts.)

Now… onto a few significant “minuses.”

In a New York Times article titled “A Warning on Nutmeg,” the author points us to the historical fact that, “[i]n the Middle Ages, it was used to end unwanted pregnancies.”[44]

This past employment as an herbal abortifacient would probably be little more than a footnote, were it not for the fact, reported by the Journal of Medical Toxicology, that there is such a thing as “nutmeg poisoning.”[45]

And this leads us to the somewhat darker side of nutmeg’s properties: myristicin’s potentially toxic effects.[46]

The concern shouldn’t be overstated, however. These poisoning cases are rare. They tend to involve teenagers horsing around. So, you don’t necessarily have to worry – for example, if you’re following quantity information in a tried-and-true recipe.

Still, interested persons should proceed with caution, since there’s little to go on in terms of precise information regarding how much nutmeg may be needed to cause some of the nastier effects (like hallucinations, nausea, and vomiting).

The New York Times says: “It takes a fair amount of nutmeg — two tablespoons or more — before people start exhibiting symptoms.”[47] That quantities of this sort (two tablespoons or more) are required is underscored by some of the poisoning reports available.[48]

Healthline suggests that doses can be less and still result in adverse events. In the article “High on Nutmeg,” writer Eleesha Lockett tell us: “According to the case studies from the Illinois Poison Center, even 10 grams (approximately 2 teaspoons) of nutmeg is enough to cause symptoms of toxicity. At doses of 50 grams or more, those symptoms become more severe. Like any other drugs, the dangers of nutmeg overdose can occur no matter the method of delivery.”[49]

(For more on this, see my YouTube video, HERE.)

Just handle nutmeg with care. For example, use only as directed by trusted recipes, and keep it out of reach of kids, teenagers, and the cognitively impaired.[50]

Spice #12: Oregano (Origanum vulgare)

Oregano’s Antioxidant Profile

“Oregano is rich in antioxidants…”.[51]

A glance at my “profile” for oregano reveals a plethora of powerful, constituent antioxidants, including rosmarinic acid – a compound also found in lemon balm (Melissa officinalis), peppermint (Mentha × piperita), rosemary (Rosmarinus officinalis), sage (Salvia officinalis), and thyme (Thymus vulgaris) – and which, in addition to its antioxidant capabilities, “…possesses many biological activities including …anti-inflammatory, anticancer, antiviral, antibacterial, and neuroprotective effects.”[52]

“Dietary intake of oregano oil has been reported to significantly delay lipid oxidation in animal models…”.[53]

One Science Daily post humorously puts things this way: “In what may be good news for pizza lovers and Italian-food connoisseurs everywhere, the herbs with the highest antioxidant activity belonged to the oregano family. In general, oregano had 3 to 20 times higher antioxidant activity than the other herbs studied,” according to at least one American-Chemical-Society investigation.[54]

Spice #13: Rosemary (Salvia rosmarinus / Rosmarinus officinalis)

Rosemary’s Antioxidant Profile

Rosemary has “potent antioxidant properties” which “have been mainly attributed to its major diterpenes, carnosol and carnosic acid, as well as to the essential oil components”[55] – names for some of which you can read on the “dossier” that I have prepared.

We looked at another, characteristic antioxidant component – namely, rosmarinic acid – when we covered oregano, above.

Rosemary is quite noteworthy. In fact, I have two video presentations dedicated to it (viewable HERE and HERE), including one (ßthe second link!) where I (unofficially!) name it my pick for the second-best “Alzheimer’s herb” – just behind Gingko biloba.

Suffice it to say that rosemary is one of those “powerhouse” herbs that appears to be capable of attacking Alzheimer’s from multiple angles, including: providing “…general antioxidant-mediated neuronal protection,” guarding against “brain inflammation,” and even possibly hindering “amyloid-beta (Aβ) formation.”[56]

(Recall that nutmeg was alleged to have a similar, Aβ-inhibiting action. For a review, see the relevant section, above. And, for similar remarks about sage, continue, below!)

Spice #14: Sage (Salvia officinalis)

Sage’s Antioxidant Profile

As Healthline puts it: Sage is “Loaded With Antioxidants”![57] In fact, the herb reportedly “…contains over 160 distinct polyphenols, which are plant-based …antioxidants…”.[58]

Like oregano and rosemary before it, sage also contains nonnegligible quantities of rosmarinic acid (see the writeup on oregano for details). But it also has salvianolic acid, a chemical that is somewhat unique to Salvia plants.

The widely studied Red-Sage species, Salvia miltiorrhiza, for example, has “Salvianolic acid B (Sal B),” a “major and …active anti-oxidant …[that] protects diverse kinds of cells from damage caused by a variety of toxic stimuli.”[59]

Though, I hasten to add that “…salvianolic acid” shows up as one of the “major components” in “…analyzed samples of S. officinalis…,” or garden-variety sage, as well.

The remarks made in one scientific article are worth quoting at length.

“Amongst many herbal extracts, Salvia species are known for the beneficial effects on memory disorders… S. officinalis (common sage), Salvia lavandulaefolia (Spanish sage), and Salvia miltiorrhiza (Chinese sage) have been used for centuries as restoratives of lost or declining mental functions such as in Alzheimer’s disease (AD).

“…In AD, the enzyme acetyl cholinesterase (AChE) is responsible for degrading and inactivating acetylcholine, which is a neurotransmitter substance involved in the signal transferring between the synapses. AChE inhibitor drugs act by counteracting the acetylcholine deficit and enhancing the acetylcholine in the brain. …Essential oil of S. officinalis has been shown to inhibit 46% of AChE activity at a concentration of 0.5 mg/ml.

“…A study shows that S. officinalis improves the memory and cognition… A randomized, double-blind clinical study has shown that an ethanolic extract from common sage (S. officinalis) as well as Spanish sage (S. lavandulaefolia) is effective in the management of mild to moderate AD…”

“…The cytoprotective effect of sage against Aβ (amyloid-beta plaques) toxicity in neuronal cells has also been proven by …a study which provides the pharmacological basis for the traditional use of sage in the treatment of AD.”[60]

Therefore, sage – along with other plants like gingko, rosemary, and saffron – belongs high on any list of possible herbal Alzheimer’s interventions.

Let me interject, at this point, that if you want more detailed information on or about any of these herbs, then I would invite you to do a little bit of research yourself on PubMed.

First of all, many articles specify more of the antioxidant constituents than I do.

Secondly, as just illustrated by the previous, long quotation, numerous scholarly articles excavate the therapeutic potential of these spices much more completely – and expertly – than I can do in this space.

(However, for a nontechnical introduction, I invite you to check out my own treatment of sage on YouTube, HERE.)

(For a refresher regarding the significance of this activity for Alzheimer’s Disease, see the entries for black pepper and garlic, above. See also: my YouTube presentations on the function of acetylcholinesterase inhibitors, HERE; hypothetical “acetylcholinesterase deficiency,” HERE; and FDA-approved Alzheimer’s drugs which have acetylcholinesterase-inhibiting functions, HERE.)

Spice #15: Thyme (Thymus vulgaris)

Thyme’s Antioxidant Profile

Regarding thyme, one website reports: “It turns out that this useful kitchen herb is also a high-antioxidant food.”[61]

In fact, thyme – or, at least, its essential oil – is arguably one of the most potent herbal antioxidants. One set of authors reports: “The best antioxidant[62] was T. vulgaris oil.”[63]

And among thyme’s most important constituents – a summary of which you can see in the “profile” that I prepared – is thymol, itself one candidate (on a short list) for the title most powerful antioxidant.

One journal states: “Thymol, carvacrol, and eugenol are the most powerful antioxidants…”.[64]

Thymol is so potent that even “waste thyme extract can… be used as an antioxidant either in food or pharmaceutical emulsions…”.[65]

In terms of Alzheimer’s-Disease relevance, I note that “…thymol decreased the effects of Aβ on memory and could be considered as neuroprotective.”[66]

On top of all this, thyme also displays antimicrobial properties.[67]

(For more on thyme, including a segment on borneol (a constituent that is able to improve the transportation of other therapeutic compounds into the brain) see the YouTube version of this presentation, HERE.)

Spice #16: Turmeric (Curcuma longa)

Turmeric’s Antioxidant Profile

This herb is more widely known – and regarded – for its potent anti-inflammatory properties.[68]

But, make no mistake, “[t]urmeric is a powerful antioxidant,” also.[69]

The key component is something called “curcumin.” Now, the relationship between curcumin and turmeric is a bit tricky, especially when it comes to supplements. Curcumin has been thoroughly studied for its health benefits – which are numerous.

But, by itself, curcumin does not have the same health benefits from a practical standpoint, because it doesn’t get absorbed well in the digestive tract.

An article in USA Today makes the point.

“Curcumin is a nutritional compound located within the rhizome, or rootstalk, of the turmeric plant. An average turmeric rhizome is about 2% to 5% curcumin. …[I]t’s the curcumin …that has the powerful health benefits. …You would have to take hundreds of [turmeric] capsules to get a clinically studied amount of curcumin. …[But p]lain curcumin extracts are poorly absorbed in the intestinal tract.”[70]

Synergy

One possible workaround arguably depends upon the concept of synergism – mentioned earlier. Recall that this has to do with the idea of “combining” or “pooling” potencies.

So, for example, curcumin can be taken with another of turmeric’s constituents, namely, aromatic turmerone, sometimes abbreviated as “ar-turmerone.”[71]

Another possibility is a combination of curcumin and black pepper, the common spice discussed earlier in this article. “[P]iperine is the major active component of black pepper and, when combined in a complex with curcumin, has been shown to increase bioavailability by 2000%.”[72]

A further illustration of the power of synergy is offered by the Journal of Agricultural Food Chemistry, where we read the following. When sage and thyme are combined, antioxidant constituents, including “[c]arnosol, rosmanol, epirosmanol, isorosmanol, galdosol, and carnosic acid” together “exhibited remarkably strong activity, which was comparable to that of alpha-tocopherol.”[73]

“Alpha-tocopherol is the most active form of vitamin E in humans.[74] It is also a powerful biological antioxidant. Vitamin E supplements are usually sold as alpha-tocopheryl acetate, a form that protects its ability to function as an antioxidant.”[75]

Blends

And… don’t forget about spice blends! Many blends provide you with these common herbs in combination.

For instance, curry powder frequently includes ingredients such as coriander, fenugreek, ginger, and – of course – turmeric.

Chili powder might have garlic in addition to cayenne or paprika.

Italian-seasoning blends are typically going to include basil, garlic powder, oregano, Rosemary, thyme, and so on. Sometimes there’s an assist from things like marjoram or parsley – both of which I get into in part 2. (See HERE.)

There are a number of other blends, of course. For example, there’s poultry seasoning, which can have oregano, sage, and rosemary, but also secondary constituents like black pepper and marjoram.

My point in mentioning blends in these cursory comments is simply this.

Even if you look at your spice rack see discover that you don’t have most – or any – of the sixteen herbs expressly named on my list, if you have a few blends, you might find that you have more than you think you do.

Vitamin C

As a coda, I’ll add that many – in fact, nearly all – of these herbs also include vitamins. In conversations about antioxidants, one of the most significant vitamins is Vitamin C, or ascorbic acid.

Take thyme, for example. Thyme is frequently touted as a significant source of vitamin C. In its article “20 Foods That Are High in Vitamin C,” Healthline reports that “[o]ne ounce (28 grams) of fresh thyme provides 45 mg of vitamin C, which is 50% of the D[aily]V[alue].”[76]

Of course, you’re probably unlikely to consume that much thyme at one sitting.

Moreover, note that the article in question specifically mentions fresh thyme – as opposed to the dried stuff. It’s arguable that fresh herbs are often higher than their dried counterparts in terms of vitamin content, but lower in terms of other antioxidants – or, at least, in terms of measured, overall antioxidant potency.[77]

(I plan on tackling the vexed topic of “ORAC values” in another place.)

Bottom line: just be aware that many of these herbs can deliver some vitamin content. In fact, every one of the herbs that I survey, here, is reported to have some Vitamin-C content.

For More Information

Where can you go for more?

  • PubMed is a publicly searchable database of scholarly articles, many of which (though, not all) are posted in full-text format. Just use search strings such as <“antioxidant” + [your favorite herb]>. PubMed is accessible, HERE.
  • Among the numerous, informative articles that you may find is an offering like this: “Antioxidant Activity of Spices and Their Impact on Human Health: A Review.”[78]
  • That article is actually published by the international, peer-reviewed, academic journal Antioxidants – located HERE – which, as its name suggests, focuses on the topic that has occupied us in this post.
  • Additionally, though, my website and YouTube channel are sources for basic introductions to many topics in the vicinity. Some of the titles that readers may find interesting include:
    1. Antioxidants on Your Kitchen Spice Rack, Part 1” (16 Common Herbs)
    2. Antioxidants on Your Kitchen Spice Rack, Part 2” (16 Less-Common Herbs)
    3. Alzheimer’s Herbs, Part 1: “Top 10 Ayurvedic Herbs” (except for turmeric!)
    4. Alzheimer’s Herbs, Part 2: “10 Miscellaneous Brain-Health Herbs” (including sage)
    5. Alzheimer’s Herbs, Part 3: “Top 5 Herbs for Alzheimer’s Disease” (turmeric is in this one!)
    6. All 25 of the herbs in the three installments just mentioned appear in a written article on my website, available HERE.
    7. Finally, I’ll mention my YouTube presentation on “Rosemary” (a dedicated, early video I made on this spice)

[By Matthew Bell]

Notes:

[1] Basil is sometimes referred to as “Sweet Basil.”

[2] Shadi Sarahroodi, Somayyeh Esmaeili, Peyman Mikaili, Zahra Hemmati, and Yousof Saberi, “The effects of green Ocimum basilicum hydroalcoholic extract on retention and retrieval of memory in mice,” Ancient Science of Life, vol. 31, no. 4, Apr.-Jun. 2012, pp. 185-189, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3644756/>.

[3] According to: Biljana Kaurinovic, Mira Popovic, and Sanja Vlaisavljevic, “In Vitro and in Vivo Effects of Laurus nobilis L. Leaf Extracts,” Molecules, vol. 15, no. 5, May 2010, pp. 3,378-3,390, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6263372/>.

[4] For just one hint of this, see: Lokraj Subedee, R. Suresh, M. Jayanthi, H. Kalabharathi, A. Satish, and V. Pushpa, “Preventive Role of Indian Black Pepper in Animal Models of Alzheimer’s Disease,” Journal of Clinical and Diagnostic Research, vol. 9, no. 4, Apr. 2015, pp. FF01-FF04, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4437082/>.

[5] Pennapa Chonpathompikunlert, Jintanaporn Wattanathorn, and Supaporn Muchimapura, “Piperine, the main alkaloid of Thai black pepper, protects against neurodegeneration and cognitive impairment in animal model of cognitive deficit like condition of Alzheimer’s disease [sic],” Food Chem. Toxicol., vol. 48, no. 3, Mar. 2010, pp. 798-802, < https://pubmed.ncbi.nlm.nih.gov/20034530/>.

[6] BrahmaNaidu Parim, Nemani Harishankar, Meriga Balaji, Sailaja Pothana, and Ramgopal Rao Sajjalaguddam, “Effects of Piper nigrum extracts: Restorative perspectives of high-fat diet-induced changes on lipid profile, body composition, and hormones in Sprague-Dawley rats,” Pharmaceutical Biology, vol. 53, no. 9, Apr. 9, 2015, pp. 1,318-1,328, <https://pubmed.ncbi.nlm.nih.gov/25856709/>.

[7] Kazuya Murata, Shinichi Matsumura, Yuri Yoshioka, Yoshihiro Ueno, and Hideaki Matsuda, “Screening of β-secretase and acetylcholinesterase inhibitors from plant resources,” Journal of Natural Medicines, vol. 69, no. 1, Aug. 15, 2014, pp. 123-129, <https://pubmed.ncbi.nlm.nih.gov/25119528/>.

[8] J.D.D. Tamokou, et al., “Antimicrobial Activities of African Medicinal Spices and Vegetables,” Victor Kuete, ed., Medicinal Spices and Vegetables from Africa: Therapeutic Potential against Metabolic, Inflammatory, Infectious and Systemic Diseases, London: Academic Press; Elsevier, 2017, p. 223, <https://books.google.com/books?id=SHjUDAAAQBAJ&pg=223>.

[9] Cholecalciferol, also known as Vitamin D3, is a particularly highly regarded variety.

[10] Setareh Sanati, et al., “A Review of the Effects of Capsicum annuum L. and Its Constituent, Capsaicin, in Metabolic Syndrome,” Iranian Journal of Basic Medical Sciences, vol. 21, no. 5, May 2018, pp. 439-448, <https://pubmed.ncbi.nlm.nih.gov/29922422/>.

[11] “Diabetes and Alzheimer’s linked,” Mayo Clinic, May 22, 2019, <https://www.mayoclinic.org/diseases-conditions/type-2-diabetes/in-depth/diabetes-and-alzheimers/art-20046987>.

[12] Arguably, they’re literally identical. At least, in some preparations, they seem to be the same herb. But, since I am not experienced enough to know whether this is the usual state of affairs, I’ll stick to the more reserved word, and simply say that they’re similar.

[13] Lizzie Streit, “8 Science-Backed Benefits of Paprika,” Healthline, Aug. 20, 2019, <https://www.healthline.com/nutrition/paprika-benefits>.

[14] Hong-Gi Kim, et al., “Binding, Antioxidant and Anti-proliferative Properties of Bioactive Compounds of Sweet Paprika (Capsicum annuum L.),” Plant Foods for Human Nutrition, vol 71, no. 2, Jun. 2016, pp. 129-136, <https://pubmed.ncbi.nlm.nih.gov/27184000/>.

[15] F. Márkus, H. Daood, J. Kapitány, and P. Biacs, “Change in the carotenoid and antioxidant content of spice red pepper (paprika) as a function of ripening and some technological factors,” Journal of Agricultural Food Chemistry, vol. 47, no. 1, Jan. 1999, pp. 100-107, <https://pubmed.ncbi.nlm.nih.gov/10563856/>.

[16] Cinnamomum verum is sometimes designated “Ceylon Cinnamon.” It is a close cousin to the Chinese variety, Cinnamomum cassia, which is more commonly found on grocery-store shelves. According to an article in the Wall Street Journal, C. verum is considered safer than C. cassia – at least in high doses. Additionally, C. verum is assumed to share many of the same salubrious properties of C. cassia, which latter has (admittedly) been more thoroughly studied in scientific experiments. See Laura Johannes, “Little Bit of Spice for Health, but Which One? While Ceylon Cinnamon Is Milder Than Grocery-Store Variety, There Are Few Studies on Its Benefits,” Wall Street Journal, Oct. 14, 2013, <https://www.wsj.com/articles/little-bit-of-spice-for-health-but-which-one-1381786452>.

[17] Joe Leech, “10 Evidence-Based Health Benefits of Cinnamon,” Healthline, July 5, 2018, <https://www.healthline.com/nutrition/10-proven-benefits-of-cinnamon>.

[18] J. Dhuley, “Anti-Oxidant Effects of Cinnamon (Cinnamomum verum) Bark and Greater Cardamom (Amomum subulatum) Seeds in Rats Fed High-Fat Diet,” Indian Journal of Experimental Biology, vol. 37, no. 3, Mar. 1999, pp. 238-242, <https://pubmed.ncbi.nlm.nih.gov/10641152/>.

[19] Just a terminological note: Coriander and cilantro are the same plant, Coriandrum sativum. Some people, in some contexts, probably use the words “coriander” and “cilantro” as synonyms. But the way I’m using these words is this. “Coriander” refers to the seeds of the plant, whereas, “cilantro” refers to the aerial parts (leaves, etc.).

[20] Helle Wangensteen, Anne Samuelsen, and Karl Malterud, “Antioxidant activity in extracts from coriander,” Food Chemistry, vol.88, no. 2, Nov. 2004, pp. 293-297, <https://www.sciencedirect.com/science/article/abs/pii/S0308814604001219>.

[21] B. Deepa, C. Anuradha, “Antioxidant potential of Coriandrum sativum L. seed extract,” Indian Journal of Experimental Biology, vol. 49, no. 1, Jan. 2011, pp. 30-38, <https://pubmed.ncbi.nlm.nih.gov/21365993/>.

[22] For just one report on this, see: Ramit Ravona-Springer and Michal Schnaider-Beeri, “The association of diabetes and dementia and possible implications for nondiabetic populations,” Expert Review of Neurotherapeutics, vol. 11, no. 11, Nov. 2011, pp. 1,609-1,617, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3240939/>.

[23] Yurina Mima, Nobuo Izumo, Jiun-Rong Chen, Suh-Ching Yang, Megumi Furukawa, and Yasuo Watanabe, “Effects of Coriandrum sativum Seed Extract on Aging-Induced Memory Impairment in Samp8 Mice,” Nutrients, vol. 12, no. 2, Feb. 11, 2020, pp. 455ff, <https://pubmed.ncbi.nlm.nih.gov/20848667/>.

[24] Ibid.

[25] N. Thippeswamy and K. Naidu, “Antioxidant potency of cumin varieties—cumin, black cumin and bitter cumin—on antioxidant systems,” European Food Research and Technology, Jan. 12, 2005, vol. 220, pp. 472-476, <https://link.springer.com/article/10.1007/s00217-004-1087-y>.

[26] Sushruta Koppula and Dong Kug Choi, “Cuminum cyminum extract attenuates scopolamine-induced memory loss and stress-induced urinary biochemical changes in rats: a noninvasive biochemical approach,” Pharm. Biol., vol. 49, no. 7, Jul. 2011, pp. 702-708, <https://pubmed.ncbi.nlm.nih.gov/21639683/>.

[27] Ibid.

[28] Xian-Hui Li, Chun-Yan Li, Zhi-Gang Xiang, Fei Zhong, Zheng-Ying Chen, and Jiang-Ming Lu, “Allicin can reduce neuronal death and ameliorate the spatial memory impairment in Alzheimer’s disease models,” Neurosciences (Riyadh, Saudi Arabia), vol. 15, no. 4, Oct. 2010, pp. 237-243, <https://pubmed.ncbi.nlm.nih.gov/20956919/>.

[29] Suresh Kumar, “Dual inhibition of acetylcholinesterase and butyrylcholinesterase enzymes by allicin,” Indian Journal of Pharmacology, vol. 47, no. 4, Jul.-Aug. 2015, pp. 444-446, <https://pubmed.ncbi.nlm.nih.gov/26288480/>.

[30] See: Agatonovic-Kustrin, et al., <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6334595/>, op. cit.

[31] Ana L. Colín-González, Ricardo Santana, Carlos Silva-Islas, Maria Chánez-Cárdenas, Abel Santamaría, and Perla Maldonado, “The Antioxidant Mechanisms Underlying the Aged Garlic Extract- and S-Allylcysteine-Induced Protection,” Oxidative Medicine and Cellular Longevity, vol. 2012, 2012, p. 907,162, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3363007/>.

[32] Biljana Petrovska and Svetlana Cekovska, “Extracts from the history and medical properties of garlic,” Pharmacognosy Review, vol. 4, no. 7, Jan.-Jun. 2010, pp. 106-110, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3249897/>.

[33] “Health Benefits of Ginger,” WebMD, Melinda Ratini, reviewer, Nov. 6, 2020, <https://www.webmd.com/diet/ss/slideshow-health-benefits-ginger>.

[34] Nafiseh Khandouzi, Farzad Shidfar, Asadollah Rajab, Tayebeh Rahideh, Payam Hosseini, and Mohsen Mir Taherif, “The Effects of Ginger on Fasting Blood Sugar, Hemoglobin A1c, Apolipoprotein B, Apolipoprotein A-I and Malondialdehyde in Type 2 Diabetic Patients,” Iranian Journal of Pharmaceutical Research, vol. 14, no. 1, Winter 2015, pp. 131–140, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4277626/>.

[35] Naritsara Saenghong, et al., “Zingiber officinale Improves Cognitive Function of the Middle-Aged Healthy Women,” Evid. Based Complement Alternat. Med., vol. 2012, Dec. 22, 2011, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3253463/>.

[36] Bui Thanh Tung, Dang Kim Thu, Nguyen Thi Kim Thu, and Nguyen Thanh Hai, “Antioxidant and acetylcholinesterase inhibitory activities of ginger root (Zingiber officinale Roscoe) extract,” Journal Complementary and Integrative Medicine, vol. 14, no. 4, May 4, 2017, <https://pubmed.ncbi.nlm.nih.gov/29345437/>.

[37] See: Faizul Azam, Abdualrahman Amer, Abdullah Abulifa, and Mustafa Elzwawi, “Ginger components as new leads for the design and development of novel multi-targeted anti-Alzheimer’s drugs: a computational investigation,” Drug Design, Development and Therapy, vol. 8, 2014, pp. 2,045-2,059, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4211852/>.

[38] R. Rajamuruganab, N. Selvaganabathyc, S. Kumaraveld, C. Ramamurthyc, V. Sujathae, and C. Thirunavukkarasu, “Polyphenol contents and antioxidant activity of Brassica nigra (L.) Koch. leaf extract,” Natural Product Research, vol. 26, no. 23, Dec. 2012, pp. 2,208-2,210, <https://www.researchgate.net/profile/Dr_Chinnasamy_Thirunavukkarasu/publication/51818527_Polyphenol_contents_and_antioxidant_activity_of_Brassica_nigra_L_Koch_leaf_extract/links/55ca502608aeca747d69e63f/Polyphenol-contents-and-antioxidant-activity-of-Brassica-nigra-L-Koch-leaf-extract.pdf>.

[39] Alina Petre, “Is Mustard Good for You?” Healthline, Jan. 10, 2020, <https://www.healthline.com/nutrition/is-mustard-good-for-you>.

[40] Somayeh Kouhestani, Adele Jafari, and Parvin Babaei, “Kaempferol attenuates cognitive deficit via regulating oxidative stress and neuroinflammation in an ovariectomized rat model of sporadic dementia,” Neural. Regen. Res., vol. 13, no. 10, Oct. 2018, pp. 1,827-1,832, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6128063/>.

[41] Jillian Kubala, “8 Science-Backed Benefits of Nutmeg,” Healthline, Jun. 12, 2019, <https://www.healthline.com/nutrition/nutmeg-benefits>.

[42] Ibid.

[43] Milad Iranshahy and Behjat Javadi, “Diet therapy for the treatment of Alzheimer’s disease in view of traditional Persian medicine: A review,” Iranian Journal of Basic Medical Sciences, vol. 22, no. 10, Oct. 2019, pp. 1,102-1,117, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6885391/>.

[44] Deborah Blum, “A Warning on Nutmeg,” New York Times, Nov. 25, 2014, <https://well.blogs.nytimes.com/2014/11/25/a-warning-on-nutmeg/>.

[45] See: Jamie Ehrenpreis, Carol DesLauriers, Patrick Lank, P. Keelan Armstrong, and Jerrold Leikin, “Nutmeg Poisonings: A Retrospective Review of 10-Years Experience from the Illinois Poison Center, 2001–2011,” J. Med. Toxicol., vol. 10, no. 2, Jun. 2014, pp. 148-151, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4057546/>.

[46] Note: Beside the fact that “[m]yristicin is present in nutmeg” – and the related “mace” – it’s also present in “…black pepper, parsley, celery, dill, and carrots.” This is according to the chapter titled “Toxins in Food: Naturally Occurring,” by D. Hwang and T. Chen, contributors to the academic volume Encyclopedia of Food and Health (Oxford and Waltham, Mass: Academic Press; Elsevier, 2016), edited by Benjamin Caballero, Paul Finglas, and Fidel Toldrá (text excerpted at <https://www.sciencedirect.com/topics/pharmacology-toxicology-and-pharmaceutical-science/myristicin>). However, I am reporting on myristicin in relation to nutmeg – and not in relation to other of the named plants – because the quantities are orders of magnitude higher in nutmeg, resulting in the fact that “…nutmeg and mace induce greater narcotic and psychotomimetic activity than” some other herbs, or even of “…an equivalent amount of myristicin or elemicin, also a component of nutmeg” separately. Ibid.

[47] Blum, loc. cit.

[48] Ehrenpreis, et al., op. cit. In fact, one report involved “…ten tablespoons of nutmeg.” Ibid.

[49] Eleesha Lockett, “Can You Get High on Nutmeg? Why This Isn’t a Good Idea,” Gerhard Whitworth, reviewer, Healthline, Aug. 31, 2018, <https://www.healthline.com/health/high-on-nutmeg>. Note that in a Google snippet, the article’s title displays as “High on Nutmeg: The Effects of Too Much and the Dangers”; whereas, on the actual Healthline website, the title reads “Can You Get High on Nutmeg? Why This Isn’t a Good Idea.” Presumably, the difference has to do with Search-Engine-Optimization (SEO) settings, which is an esoteric conversation that would implicate technical terms like “metadata” and “metatags,” and lies far afield from anything I’ll be delving into, presently.

[50] Of course, the focus of my work is on people with Alzheimer’s Disease and other forms of dementia. These conditions negatively affect memory and reasoning. Confused sufferers can sometimes expose themselves (or others) to dangers – whether advertently or inadvertently. For instance, one journal article reports on the case of one woman whose cognitively afflicted “…husband put nutmeg on his steak instead of pepper.” Els van Wijngaarden, et al., “Entangled in uncertainty: The experience of living with dementia from the perspective of family caregivers,” PLoS One (Public Library of Science), vol. 13, no. 6, Jun. 13, 2018, p. e0198034, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5999274/>.

[51] Rachael Link, “6 Science-Based Health Benefits of Oregano,” Healthline, Oct. 27, 2017, <https://www.healthline.com/nutrition/6-oregano-benefits>.

[52] Niloufar Ansari and Fariba Khodagholi, “Natural Products as Promising Drug Candidates for the Treatment of Alzheimer’s Disease: Molecular Mechanism Aspect,” Current Neuropharmacology, vol. 11, no. 4, Jul. 2013, pp. 414-429, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3744904/>.

[53] Muhammad Ayaz, Abdul Sadiq, Muhammad Junaid, Farhat Ullah, Fazal Subhan, and Jawad Ahmed, “Neuroprotective and Anti-Aging Potentials of Essential Oils from Aromatic and Medicinal Plants,” Frontiers in Aging Neuroscience, vol. 9, May 30, 2017, p. 168, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5447774/>.

[54] “Researchers Call Herbs Rich Source of Healthy Antioxidants; Oregano Ranks Highest,” Science Daily, Jan. 8, 2002, <https://www.sciencedaily.com/releases/2002/01/020108075158.htm>; citing: American Chemical Society. On oregano outperforming other herbals in terms of its antioxidant abilities, see also: Snezana Agatonovic-Kustrin, Ella Kustrin, and David Morton, “Essential oils and functional herbs for healthy aging,” Neural Regeneration Research, vol. 14, no. 3, Mar. 2019, pp. 441-445, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6334595/>.

[55] Aleksandar Rašković, Isidora Milanović, Nebojša Pavlović, Tatjana Ćebović, Saša Vukmirović, and Momir Mikov, “Antioxidant activity of rosemary (Rosmarinus officinalis L.) essential oil and its hepatoprotective potential,” BMC Complementary and Alternative Medicines (alternatively titled BMC Complementary Medicine and Therapies), vol. 14, Jul. 7, 2014, p. 225, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4227022/>.

[56] As discussed in: Solomon Habtemariam, “The Therapeutic Potential of Rosemary (Rosmarinus officinalis) Diterpenes for Alzheimer’s Disease,” Evidence Based Complementary and Alternative Medicine, vol. 2016; Jan. 28, 2016, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4749867/>.

[57] Ryan Raman, “12 Health Benefits and Uses of Sage,” Healthline, Dec. 14, 2018, <https://www.healthline.com/nutrition/sage>.

[58] Ibid.

[59] Yan-Hua Lin, Ai-Hua Liu, Hong-Li Wu, Christel Westenbroek, Qian-Liu Song, He-Ming Yu, Gert Horst, and Xue-Jun Li, “Salvianolic acid B, an antioxidant from Salvia miltiorrhiza, prevents Abeta(25-35)-induced reduction in BPRP in PC12 cells,” Biochemical and Biophysical Research Communications, vol. 348, no. 2, Jul. 28, 2006 [online], Sept. 22, 2006 [print], pp. 593-609, <https://pubmed.ncbi.nlm.nih.gov/16890202/>.

[60] Mohsen Hamidpour, Rafie Hamidpour, Soheila Hamidpour, and Mina Shahlari, “Chemistry, Pharmacology, and Medicinal Property of Sage (Salvia) to Prevent and Cure Illnesses such as Obesity, Diabetes, Depression, Dementia, Lupus, Autism, Heart Disease, and Cancer,” Journal of Traditional and Complementary Medicine, vol. 4, no. 2, Apr.-Jun. 2014, pp. 82-88, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4003706/>.

[61] Amanda Rose, “Antioxidants in Thyme,” Traditional Foods; The Antioxidant Project, Jun. 30, 2011, <http://www.traditional-foods.com/antioxidants/thyme/>.

[62] At least, it was the best among the five explicitly tested: bitter orange (Citrus aurantium), Fennel (Foeniculum vulgare), Mediterranean cypress (Cupressus sempervirens), Tasmanian blue gum (Eucalyptus globulus), and Thyme (Thymus vulgaris).

[63] Smail Aazza, Badiâ Lyoussi, and Maria Miguel, “Antioxidant and antiacetylcholinesterase activities of some commercial essential oils and their major compounds,” Molecules, vol. 16, no. 9, Sept. 7, 2011, pp. 7,672-7,690, <https://pubmed.ncbi.nlm.nih.gov/21900869/>.

[64] Yasiel Crespo, Luis Sánchez, Yudel Quintana, Andrea Cabrera, Abdel del Sol, and Dorys Mayanchaa, “Evaluation of the synergistic effects of antioxidant activity on mixtures of the essential oil from Apium graveolens L., Thymus vulgaris L. and Coriandrum sativum L. using simplex-lattice design,” Heliyon, Jun. 15, 2019, vol. 5, no. 6, p. e01942, <https://pubmed.ncbi.nlm.nih.gov/31245650/>.

[65] Soukaïna El-Guendouz, Smail Aazza, Susana Dandlen, Nessrine Majdoub, Badiaa Lyoussi, Sara Raposo, Maria Antunes, Vera Gomes, and Maria Miguel, “Antioxidant Activity of Thyme Waste Extract in O/W Emulsions,” Antioxidants (Basel, Switzerland), vol. 8, no. 8, Jul. 25, 2019[online], Aug. 2019 [print], pp. 243, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6719112/>.

[66] Masoumeh Asadbegi, Parichehreh Yaghmaei, Iraj Salehi, Alireza Komaki, Azadeh Ebrahim-Habibi, “Investigation of thymol effect on learning and memory impairment induced by intrahippocampal injection of amyloid beta peptide in high fat diet- fed rats,” Metabolic Brain Disorder, vol. 32, no. 3, Mar. 2, 2017 [online], Jun. 2017, [print], pp. 827-839, <https://pubmed.ncbi.nlm.nih.gov/28255862/>.

[67] Monika Sienkiewicz, Monika Łysakowska, Paweł Denys, and Edward Kowalczyk, “The antimicrobial activity of thyme essential oil against multidrug resistant clinical bacterial strains,” Microbial Drug Resistance, vol. 18, no. 2, Nov. 21, 2011 [online], Apr. 2012 [print], pp. 137-148, <https://pubmed.ncbi.nlm.nih.gov/22103288/>.

[68] Julie Jurenka, “Anti-inflammatory properties of curcumin, a major constituent of Curcuma longa: a review of preclinical and clinical research,” Alternative Medicine Review, vol. 14, no. 2, Jun. 2009, pp. 141-153, <https://pubmed.ncbi.nlm.nih.gov/19594223/>.

[69] “The health benefits of turmeric,” Nuffield Health, Nov. 18, 2020, <https://www.nuffieldhealth.com/article/the-health-benefits-of-turmeric>.

[70] Terry Naturally, “The ways turmeric and curcumin differ might surprise you,” USA Today, May 1, 2019, <https://www.usatoday.com/story/sponsor-story/terry-naturally/2019/05/01/ways-turmeric-and-curcumin-differ-might-surprise-you/3541923002/>.

[71] Ibid.

[72] Susan Hewlings and Douglas Kalman, “Curcumin: A Review of Its’ Effects on Human Health,” Foods, vol. 6, no. 10, Oct. 22, 2017, p. 92, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5664031/>. A similar extract is sometimes referred to as “bioperine.”

[73] Kayoko Miura, Hiroe Kikuzaki, and Nobuji Nakatani, “Antioxidant activity of chemical components from sage (Salvia officinalis L.) and thyme (Thymus vulgaris L.) measured by the oil stability index method,” J. Agric. Food Chem., vol. 50, no. 7, Mar. 27, 2002, pp. 1,845-1851, <https://pubmed.ncbi.nlm.nih.gov/11902922/>.

[74] Though, see my YouTube-video presentation Antioxidants, Part 2 to discover gamma-tocopherol, a form of Vitamin E more commonly found in seeds – such as sesame seeds, which are on my list.

[75] M. Saljoughian, “Natural Powerful Antioxidants,” U.S. Pharmacist, vol. 1, Jan. 23, 2007, p. HS38-HS42, <https://www.uspharmacist.com/article/natural-powerful-antioxidants>.

[76] Caroline Hill, Jun. 5, 2018, <https://www.healthline.com/nutrition/vitamin-c-foods>.

[77] Also, pasteurization or processing can cause the vitamin content (especially in the case of Vitamin C) to diminish.

[78] Alexander Yashin, Yakov Yashin, Xiaoyan Xia, and Boris Nemzer, Antioxidants (Basel, Switzerland), vol. 6, no. 3, Sept. 15, 2017, p. 70, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5618098/>. Innumerable other articles could be given at this point, including many cited elsewhere in this post, or in any of the companion videos, but also: T. Alan Jiang, “Health Benefits of Culinary Herbs and Spices,” Journal of AOAC International, vol. 102, no. 2, Jan. 16, 2019 [online], Mar. 1, 2019 [print], pp. 395-411, <https://pubmed.ncbi.nlm.nih.gov/30651162/>.

Intro to Medicaid: Medicaid Vs. Medicare; Long-Term Care

My dad Jim died of Alzheimer’s disease in 2016.

In order to help pay for my dad’s nursing home care, my mom had to reduce her already modest assets as part of what is commonly referred to as a Medicaid spend-down.

But what is Medicaid? How do you qualify for it? Who is it designed to assist? And why do so many people have to expend their life savings in order to get it?

Let’s take a look at Medicaid.

In this introductory article, I’ll lay some basic groundwork – and provide a few essential definitions and concepts.

In two follow-up articles, we’ll discuss the all-important financial qualifications: first assets, then income. So… stay tuned!

(Also, be aware that this additional content is already available in the form of YouTube presentations and can be accessed HERE and HERE.)

Disclaimers

First of all, I need to give the usual disclaimers and in fact I want to amplify what I normally stay. I am not an attorney. I am not a financial advisor of any kind. You can consider the topics that I discuss to be research leads just to help you as you are thinking through these issues and hopefully help to shorten your learning curve in some of these important topics. But nothing that I say in this video should be construed as advice of any kind. If you need actual recommendations and evaluations, you need to consult with licensed professionals in your area. In this case, I am talking about attorneys who are knowledgeable about Medicaid, or other Medicaid experts.

The usual disclaimers are especially important in this case. That’s because Medicaid is just so complex; Medicaid’s rules and regulations are subject to amendment and modification; eligibility and other limits can be expected to change almost annually; Medicaid has as many different variations as there are US states.

So, you should meet with someone, not just is knowledgeable about the rough contours of the entire program, but somebody who knows the nitty-gritty of your local situation or state situation and how best to advise you.

What is Medicaid?

Medicaid is a United States government program that is designed to help impoverished people pay their medical expenses.

Sometimes, Medicaid is referred to as a joint federal and state program. Other times, Medicaid is referred to as a federal program that is administered by the U.S. states. And, sometimes, it is thought of mainly as a state program that has infusions of federal cash.

The federal government generally provides funding for benefits that are mandated at the federal level, which makes sense. The states, in turn, provide administrative services. They also pay for state-specific benefits.

However you think of it, or by whatever name you call it, Medicaid has a foot on the federal level government as well as on the state level.

This is one key reason for its (aforementioned) complexity. Namely, the particulars of how the plan is implemented and managed are going to vary according to the state that you are in.

Medicaid Isn’t Only for Long-Term Care

It is important to understand at the outset that Medicaid is a broad program and can cover many different medical services.

For example, it can cover routine doctor visits. It can cover hospital visits, long-term-care facilities, nursing homes and the like of that.

The long-term-care applications will be my focus in this series (since it’s the obvious concentration of the website). But Medicaid can cover a lot more than that.

And if you have need for additional services, for yourself or for your family, I am sure there are other resources that will expand my treatment.

Potential Pitfalls

Because of the complexity of the topic, there are certainly going to be a number of pitfalls.

Firstly, it’s easy to confuse Medicaid with Medicare. Not only are the two words similar, but both programs are overseen by the same government agency, the Centers for Medicare and Medicaid services, or CMS. CMS is a major division of the United States Executive Branch Department of Health and Human Services, with the cabinet-level Secretary of Health and Human Services at the helm of both programs.

However, these programs differ in terms of who is eligible and what services are covered.

Medicare is basically health insurance for seniors. Some of the premiums might vary in terms of the amount that is paid. But, otherwise, Medicare covers seniors of any income level.

Here, “senior” simply means a person who is 65 years of age or older.

There is an important caveat.

Medicare is called upon to deliver health coverage to people of any age, provided that they also are recipients of Social Security Disability Income (or SSDI), or if they suffer from end-stage renal failure (or kidney disease). Medicare itself is complex.

Medicare is a complex program itself. For my own YouTube-video treatment, see HERE.

In broad strokes, though, Medicare Parts A and B cover hospital and doctor visits, respectively. Part D was added to help provide prescription-drug coverage.

You can talk about something referred to as Medicare “Part C,” also known as “Medicare Advantage” plans. (These function something like Medicare HMOs)

And, to complicate matters further, Medicare Parts A, B and D can be further augmented with what are called “Medicare Supplement” (or “Medigap”) plans. (These, in turn, are also designated with letters like “Medicare-Supplement A,” “Medicare-Supplement F,” and so on.)

Medicare & Social Security

Historically, Medicare has a fairly tight relationship with Social Security.

For example, although it does have a deductible, Medicare Part A is usually delivered premium-free to people who are fully qualified to receive Social Security benefits. It is in this sense that Medicare Part A is often referred to as “automatic.”

It is important to note that if you are not eligible to receive Part A cost free, you may be able to purchase it.

Medicare has long had this close association with the Social-Security program

In fact, Medicare’s eligibility age, 65, once corresponded to the Full Retirement Age (or “FRA”)[1] for Social Security recipients. Even though the full-retirement age has been increased for most retirees, Medicare’s eligibility age has remained the same.

Medicare part B is never free. Though it’s actual premium cost will depend on many factors, including your income level.

Medicaid Will Be Our Focus

As stated from the outset, going forward, this series will be concerned with Medicaid (and its financial requirements), the government’s financial-assistance program for impoverished people – regardless of their age.

So, we come to one quick way of distinguishing Medicare and Medicaid.

Medicare has an age requirement,[2] but no income requirement. As long as you are 65 years of age, or older, you are eligible for Medicare.

On the other hand, Medicaid has no age requirement, but it does have an income requirement.

To rephrase: Medicare covers people 65+ years of age, of any income level; Medicaid covers any aged person, but only if they are of a low-income level.

Which Program Covers Long-Term-Care Costs?

“Medicaid also covers long-term care costs, both in a nursing home and at-home care. Medicare does not provide this coverage.”[3]

As noted by CNN, long-term care and nursing home costs are not generally covered by Medicare.

Medicare really only covers “curative,” skilled-nursing care – in a Medicare-approved facility. And, even then, it only covers skilled-nursing-home costs, provided that your entry into a qualifying facility followed a hospital stay of at least three days.

Additionally, Medicare coverage is calibrated to what are termed “episodes of care.” And each skilled-nursing-home visit is only covered up to 100 days per episode.

So, since Alzheimer’s and related dementias are not “episodic” in the relevant sense, we can basically say that Medicare is going to be irrelevant for the purposes of long-term care and nursing home care.

Another reason for this irrelevance is that long-term care is often bound up with what is called custodial care, and this is to be distinguished from skilled nursing care.

Basically, custodial care is help with the activities of daily living, whereas skilled nursing care is what you would normally think of in terms of medical care, medical testing, the administration of drugs, and the like.

(For my YouTube presentation explaining the difference between custodial and skilled nursing care, see HERE. For my written article on the same topic, see HERE.)

As if things weren’t confusing enough as it is, Medicaid is able to pay some of the expenses associated with Medicare. Examples of this would include the Parts B and D premiums, the Part-A premium (when it is applicable), and the various Medicare “co-pays” and deductibles.

Intro to Medicaid Eligibility

Each state sets its own guidelines for eligibility, within various limits.

For many states, Medicaid assistance is available to people who are below, at, or near the federal poverty level, or FPL.

Recipients cannot have incomes that exceed about 130-140% of the FPL, adjusted for household size. And these low-income requirements are very strict.

In addition, Medicaid recipients are not allowed to have much by way of assets or, in the Medicaid terminology, “resources.”

But, these two financial tests – one pertaining to assets, and one pertaining to income – are quite intricate. So much so, in fact, that I will be devoting a separate article to each of these.

So, I invite you to stay tuned for those posts.

And, in the meantime, feel free to view the companion YouTube videos. My discussion of Medicaid’s asset requirements can be found HERE, and my coverage of the relevant income requirements is HERE.

Notes:

[1] Note that, in some literature, this is instead termed the “Normal Retirement Age,” or NRA. Presumably, FRA is more common due to the fact that the letters “NRA” more commonly designate the National Rifle Association.

[2] For the exceptions to this, see the previous section.

[3] “What Does Medicaid Cover?” CNN, <https://money.cnn.com/retirement/guide/insurance_health.moneymag/index21.htm>.

New Alzheimer’s ‘Blood Test’ Detects Amyloid Deposits

Alzheimer's Blood Test

What Is the New Test?

A news item crossed my radar screen a couple weeks ago. This actually goes all the way back to August 2019, and was published in the Neurology, which is an academic journal, and subsequently was picked up by various news outlets.

I saw this through a press release from Washington University School of Medicine which is located in St. Louis, Missouri. The report focused on a new blood test that’s available for Alzheimer’s Disease.

Currently, it’s in testing phases. But, it has been developed, and researchers think that it holds great promise, and let me explain why that is.

So, first of all:

What does the test do?

I have both an ARTICLE and a YouTube VIDEO on some of the possible causes for Alzheimer’s disease. One of the causes that’s often referenced is the accumulation, in the brain, of various protein “gunk.”

There are two different types of protein that are implicated in this. The first is called beta-amyloid and the second is called tau. The two of these, together, account for the various “plaques and tangles” that Alzheimer’s patients’ brains seem to be riddled with.

The 100% definitive test for this is going to be an autopsy. (For these assortment of Alzheimer’s “tests,” see – again – either my other written work, e.g., HERE, or the companion, video presentation, HERE.)

There is very little use, obviously, for an autopsy to play in terms of preventative medicine or even in treatment, while somebody’s alive. It can give you the accurate cause of death only after the fact. But it’s not going to be too good for medicinal purposes.

Now comes a blood test that promises, or at leads holds the promise, of being able to detect levels of beta-amyloid in the blood during a person’s life.

Two kinds of beta-amyloid protein are being focused upon. And those are designated “Number 40” and “Number 42.”

My layman’s “take” on this is that there is an array of these beta-amyloid’s, but that 40 and 42 somehow play particularly important roles in the pathological accumulation of these deposits in an Alzheimer’s-afflicted brain.

I should say the test has actually been going on for couple years.

What researchers have hypothesized, and has since been borne out – at least, in early stages – is that levels of beta-amyloid in the blood suggest an accumulation of the beta-amyloid in the brain.

 Why is the test important?

I mentioned that autopsies are the definitive test for Alzheimer’s. But, of course, autopsies are no good for diagnostic purposes so long as the patient is alive.

So, the most accurate test a living person can get right now is a so-called “Pet Scan,” which is shorthand for the more forbidding “positron-emission-tomography scan.”

A pet scan is essentially the best test that’s available.

But… pet scans are both expensive and time-consuming to perform. Therefore, this blood test holds out the hope for being able to be administered on a much wider scale and at far less of a cost.

Those are two points in its favor.

In theory, a blood test of this kind would be able to be given in a doctor’s office – instead of, for example, having to send the patient to an imaging center.

How Accurate Is the Blood Test?

What researchers discovered, when they compared the blood-test results against the pet-scan results, was that the blood test was 88% correlated with the pet scan.

What this seems to suggest is that, if for every 100 pet scans displaying evidence of Alzheimer’s, a blood test would detect Alzheimer’s in 88 of those cases.

At least… that’s the way I’m reading the article. (Disclaimer: I am not a medical professional! This is simply my untutored commentary on the situation. For advice, seek a healthcare expert who is familiar with your personal history.)

So, the test was (provisionally) determined to be 88% effective, in the above respect.

What Other Risk Factors Were Considered?

It’s worth pointing out that when they coupled the blood test with a couple of other risk factors – for example, age – the effectiveness increased.

It’s widely accepted that a person’s Alzheimer’s risk goes up with age. In fact, it’s said that your risk actually doubles every five years after age 65.[1]

Also, they factored in genetic predisposition. So, if you have the APO-E4 variant gene in your genetic makeup – as I myself do (see my video testimony, HERE) – then you have a higher (3 to 5 times higher actually!) chance of developing Alzheimer’s sometime in your life.[2]

Another risk factor – believe it or not – is sex. We talked about this in other places. (My AlzheimersProof.com post is HERE and my video is HERE.) Sex may not be its own risk factor. It may simply be that women live longer and therefore are more likely to manifest symptoms of Alzheimer’s and other dementias because of their longevity. But it is sometimes said that two out of three Alzheimer’s sufferers are female.[3]

When researchers incorporated age and genetic predisposition, they found that the test was 94% effective in terms of matching up to the pet scan.

What’s the Practical Benefit of an Alzheimer’s Blood Test?

But what they found, in addition to that, was that when some of the blood tests came up positive for people who had not yest been pet scanned, those people eventually came up positive on the pet scan as well.

This suggested to researchers that the test might actually be even more accurate than the pet scan, and able to detect Alzheimer’s in its very earliest stages.

Recall, also, that Alzheimer’s disease manifests in symptoms. But researchers believe that the underlying brain changes may take place as far back as a decade or more prior to the onset of symptoms.

One of the benefits obviously of early detection is they want to be able to treat people in presymptomatic stages. They feel like once memory changes, and other changes manifest in the brain, then that brain is already so damaged that it is basically beyond help.

In possibly being able to find people in the earliest stages of Alzheimer’s, another potential benefit of the blood test is that it might help with constructing clinical trials for developing interventions.

An early version of this test goes back to 2017 or even 2016. At that time, some of the articles referenced “autoimmune antibodies,” also called “autoantibodies.” The idea was that the immune system’s response to the accumulation of beta-amyloid protein might be able to be gauged. But, upon closer inspection of these articles now, in retrospect, they also seem to be describing a blood test that focuses on some of these beta-amyloid protein variants.

The fundamental idea is to try to catch Alzheimer’s disease in its very earliest stages.

Sometimes this “preclinical” stage is referred to variously as Very Mild Cognitive Impairment (especially on the so-called 7-Stage-Dementia view – about which, see HERE and HERE) or Mild Cognitive Impairment, often better known simply by the abbreviation “MCI.”

Note, however, that not all cases of MCI develop into Alzheimer’s disease. For one thing, there are other sorts of dementia (e.g., Lewy-Body Dementia, Multi-Infarct Dementia, Parkinson’s Disease, etc.) and there are traumatic brain injuries and other conditions that also might be prefaced by mild cognitive impairment. (For the difference between “Alzheimer’s Disease” and “dementia,” see my video, HERE.)

“Mild Cognitive Impairment” is, in a sense, just a catchall phrase for a certain level of mental diminishment as well as for the impoverishment of other perceptual and reasoning capabilities.

Concluding Reflection

There’s little doubt, though, that this blood test is fascinating. Even though it still has some way to go before you can expect it at a physician’s office near you, it holds promise.

Number one, it’s less expensive than the pet scan. Number two, it is able to be administered more widely, and in more environments – like doctors’ offices, and other places (such as out-patient clinics and “urgent-care” centers) – whereas the pet scan is obviously limited to an imaging center. Number three, it’s possibly more accurate than the pet scan. And, number four, it’s certainly superior to the autopsy in the sense that it holds out promise for being able to help treat and diagnose people while they are still alive – which is, quite obviously, advantageous.

Notes:

(Featured image credit: Billy W., <https://images.freeimages.com/images/large-previews/97a/blood-test-1545982.jpg>.)

[1] See: “Causes and Risk Factors for Alzheimer’s Disease,” Alzheimer’s Association, <https://www.alz.org/alzheimers-dementia/what-is-alzheimers/causes-and-risk-factors>.

[2] Though, the risk really ramps up after age 75. See the video for details.

[3] Again, see the Alzheimer’s Association: “Women and Alzheimer’s,” <https://www.alz.org/alzheimers-dementia/what-is-alzheimers/women-and-alzheimer-s>.

Intro to Family Councils in Long-Term-Care Facilities

People can end up in nursing-home and other long-term-care environments for any of several reasons. And residents may run the gamut in terms of their levels of awareness and disability.

When a resident retains his or her reason, then – in principle – he or she can “advocate” for themselves in the sense of communicating their desires and needs to facility administrators and staff. But what about cognitively impaired residents, such as those with Alzheimer’s Disease or some other form of dementia?

Such persons may require additional assistance. And this is where a Family Council can come in.

Long-Term-Care Resident Groups

Residents in long-term-care facilities, and families of people residing in such facilities, have the ability to form groups. These groups are often organized to facilitate discussions focused on resident needs and facility changes that may have to do with quality-of-care and quality-of-life issues.

As it happens, these groups, when formed, have tended to be referred to as “councils.” There are two main types of these councils that may be created: family councils and resident councils.

Resident Councils

The most basic variety of long-term-care group is going to be the resident council.

In general terms, a “resident council” is an association of individuals who live in a particular care facility and who wish to work together for the enhancement of their shared living experience.

When they exist, resident councils are independent of the care facility. To put it another way, the resident councils are not controlled or run by the facility administrators or staff.

I say that this sort of group is the “most basic” because it is natural for facility residents to provide feedback and input that pertains to their living conditions. They are interested in the facility, not simply in a psychological sense, but in an economic sense – that is, they have an interest in the policies and procedures of the relevant facility.

Family Councils

A related type of long-term-care group is the family council. As the name implies, “family councils” are groups organized and run by families (or close friends or representatives) of nursing home residents instead of the residents themselves. These family members then join with other families and speak for residents in advocating for policy and procedure changes that improve daily care and quality of life.

It should be said that nothing prevents residents from joining or participating in family councils.

Relatedly, I note that the point of the family council is not to compete with, replace, or supplant the resident council.

Rather, the point is to offer assistance to residents. And, the focus of this website being what it is – namely, Alzheimer’s Disease – it is important to observe that this assistance is especially necessary and valuable in cases where residents lack the ability to speak for themselves because of cognitive impairment, dementia, or similar conditions. (More on this, below.)

Brief Council-Related Questions and Answers (Q&A)

What Do Councils Do?

Both family and resident councils may bring to light issues surrounding care and living conditions. These may include, without limitation: the availability of food, medical equipment, silverware; the cleanliness of the facility; the functionality of things such as elevators; the reliability of internet access; and so on.

Essentially, any topic that impacts the quality of life of residents is fair game.

Once topics have been discussed, it is up to the individuals in the council as to how to proceed. Many facilities (see further on) are required to provide the council with a go-between or contact so that concerns can be delivered to the administration or staff without delay.

Councils may decide to bring their concerns to the facility orally or in writing – in a small group or via a designated council representative.

The first and preferable route would be to communicate with the facility.

However, if the facility is unreceptive for whatever reason, or if it somehow fails to satisfactorily address the expressed concerns, then councils may contemplate or undertake other actions.

Are Family Councils Only Allowed When Residents Have Dementia?

No. Family councils can be formed in order to support any person residing in a particular long-term-care facility, whether the resident is impaired or not.

However, it’s arguably even more important for a family council to be formed when resident loved ones do have cognitive impairments, since dementia sufferers may be unable to speak for themselves.

What Are Examples of Conditions That May Result in Cognitive Impairments?

Conditions may include: Alzheimer’s Disease, Creutzfeldt-Jakob Disease, Frontotemporal Dementia (Pick’s Disease), Huntington’s Chorea (Huntington’s Disease), Lewy-Body Dementia, Multi-Infarct (or Vascular) Dementia, Parkinson’s Disease, Wernicke-Korsakoff Syndrome, etc.

What If a Resident Has No Living (Or Interested) Family Members?

Residents may give permission to non-family members to participate in family councils on their behalf.[1] If a resident has a cognitive impairment, then things can get a little dicey. If you would like a non-family member to represent your interests in a family council, you may wish to record your wishes in writings prior to the manifestation of any condition that might call in question your mental fitness.

Are Long-Term-Care Facilities (Such as Nursing Homes) Required to Recognize Family Councils?

I will address this question at greater length in a forthcoming post.

But, for now, suffice it to say that Federal regulation (in Title 42 CFR 483.10) states that Medicare/Medicaid-participating nursing homes must recognize family and resident groups when they form.

Further, these facilities have to provide resident and family groups with private meeting spaces, and they need to take reasonable steps (with the approval of the groups) to make residents and family members aware of upcoming meetings in a timely manner.

Additionally, the facility must provide a designated staff person to act as a liaison to the group. This person will be responsible for providing assistance to the group and with delivering (though not necessarily writing) responses to formal requests that result from the group meetings.

Does the Long-Term-Care Facility Have to Respond to Council Requests?

The facility cannot ignore the group or suppress its ability to form. So, in this sense, yes, the facility must provide some sort of reply to a group when it is asked to do so.

This does not mean, however, that the facility is compelled to do precisely what the council requests.

Things are tricky. I will try to explore some of these issues at greater length in a future installment.

Six Characteristics of a Family Council

It may be helpful if I briefly sketch some of the qualities that a family council will have.

Autonomy

Autonomy refers to the fact that the group is self-led. Neither a family council nor a resident council is created or led by facility administrators or staff members. If the facility has started and controls a group, then it is not a family or resident council in the relevant sense – regardless of what the facility calls it.

Facilities are of course free to form their own groups; and residents or families are free to join these facility-created groups if they choose to do so. But the formation of a facility-led group is not a replacement for an autonomous family council, and joining a facility group does not bar a person from joining an autonomous group.

Facility Specificity

There may be groups that support anyone living at any nursing home. However, a family council is relative to a specific facility. The ties that bind the members of a family council have to do with the fact that members all have relatives who live in the same long-term-care facility.

Independence

Independence is an extension of autonomy. The administration does not have claims over the family council. The councils are not dependent upon the facility. Moreover, the council is not considered to be a government program. It is an independent association of families of people living at a particular facility.

Interest

A family council must be made up of a group of people who have an interest in the facility. In this context, “interest” does not simply mean a curiosity. It means there has to be an interest in more of an economic sense. Member families have to have some “stake” in the facility and, going further, in the standards according to which the facility operates.

Openness/Inclusivity

There must be no barrier to entry into the family council other than having a close friend or relative involved in the facility. The council be must inclusive or open in terms of its membership.

Privacy

The group is owed a private space that should be provided by the administration of the facility on the facility grounds. The facility cannot appoint a staff member to “sit in” on the meetings.

Administrators or staff may be invited by the group to attend meetings. But, apart from such an invitation, presumably furnished by a council officer or spokesperson, the group should be allowed to meet privately.

Once again, the facility may endorse or sponsor other, staff-led groups. And these groups may also listen to complaints or solicit feedback from families and residents. But, the existence of such facility-controlled groups does not undermine the ability of residents and families to form their own councils to advance their own interests.

Notes:

[1] Presumably, if there is a conflict, a resident could also revoke a family member’s permissions or otherwise “block” one of his or her family members from participating in a council.

Mandatory-Physician-Reporting States & Drivers’ Licensing

Does Your State Have Mandatory-Reporting Laws?

Within the United States and Canada there are laws that require a physician to report a patients with neurological or cognitive disorders to a licensing agency, as those patients may be a danger behind the wheel of a vehicle.

This type of reporting is not mandatory across the entire United States.  Currently, there are only six (6) states that mandate a physician to report on a patient’s ability to operate a motor vehicle. These six states include:

  • California
  • Delaware
  • Nevada
  • New Jersey
  • Oregon
  • Pennsylvania

Not all mandatory states direct their reporting law toward those who are afflicted with Alzheimer’s Disease or, indeed, any sort of dementia whatsoever. Below are some specifics around each mandatory state and what is defined in their law.

If you want to see and hear me present the material, feel free to watch the YouTube video that I prepared, here:

California

The state of California is the first that I will discuss. It’s a prime example of a state in which (a.) there is mandatory reporting, and (b.) that reporting is at least partially concerned with Alzheimer’s Disease expressly.

The California Legislative Information website (leginfo.legislatures.ca.gov) references chapter 3 – Disorders Characterized by Lapses of Consciousness [103900-103990].

103900 states: “Every physician and surgeon shall report immediately to the local health officer in writing, the name, date of birth, and address of every patient at least 14 years of age or older whom the physician and surgeon has diagnosed as having a case of a disorder characterized by lapses of consciousness.”

This law specifically includes Alzheimer’s Disease as a disorder that involves the requisite “lapses of consciousness.”

For additional information, see the entry on California – “What does California State Law say about Driving with Alzheimer’s?” – in my “50-State Guide to the Laws on Driving With MCI and Alzheimer’s.”

Delaware

In Delaware, the focus is on conditions that are characterized by “losses of consciousness.” In this respect, Delaware’s statutory language is similar to the “lapses of consciousness” verbiage present under California law.

Title 24 of the Delaware Code Online – Professions and Occupations Chapter 17, Medical Practice Act states: “Every physician attending or treating persons who are subject to losses of consciousness due to disease of the central nervous system shall report within 1 week to the Division of Motor Vehicles the names, ages and addresses of all such persons unless such person’s infirmity is under sufficient control to permit the person to operate a motor vehicle with safety to person and property.”

Again, for further reading, scroll down to “Delaware’s entry, HERE.

Nevada

Nevada is a good example of a state in which, while there is mandatory-physician reporting, it is not directed towards cognitive impairments such as dementia (of which, as we know, Alzheimer’s Disease is the most common variety). (On two ways of understanding the difference between Alzheimer’s Disease and dementia, see my YouTube video, HERE.)

According to the Bradley, Drendel & Jeanney law firm: “…Nevada laws can regulate the driving rights of individuals with epilepsy. In fact, doctors in our state are required to report epileptic seizures to the state’s Department of Motor Vehicles…”

You might be picking up on a recurring theme, here, but… for additional information, click down to “What does Nevada State Law say about Driving with Alzheimer’s?” in my 50-State Guide, HERE.

New Jersey

The New Jersey Academy of Ophthalmology, states, “NJ Law (N.J.S.A. 39:3-10.4) requires all physicians to report patients to the Motor Vehicle Commission within 24 hours after determining that a patient experiences any of the following:  Recurrent convulsive seizures, recurrent period of unconsciousness or impairment, or loss of motor coordination due to conditions such as, but not limited to epilepsy in any of its forms which persist or recur despite medical treatment.”

Even so, New Jersey appears to be one of those states in which physicians could potentially be held liable, legally, if they don’t report potentially dangerous drivers.

While New Jersey law has established an elective reporting system for drivers with vision deficiencies and mandatory process for reporting specific neurological dysfunctions, there is no statute that protects a physician from liability if they have failed to report a patient that may cause injuries to a third party due to a condition known by the physician.

Given this, you might think that a New Jersey doctor will err on the side of reporting. And, I may be inclined to agree.

For more info, where do you think I’ll send you? My state guide, of course! Find it, HERE.

Oregon

The Oregon Driver & Motor Vehicle Services states: “Most medical professionals are required to report drivers who can no longer drive due to impairment.”

It is required of medical professionals to report on a patient’s impairment, even if that patient has agreed to give up driving.

The Oregon Secretary of State website similarly states: “…mandatory reporting by physicians and health care providers of those persons with severe and uncontrollable cognitive or functional impairments affecting a person’s ability to safely operate a motor vehicle.”

Cognitive impairments as specified in Oregon include: attention, judgement and problem solving, reaction time, planning and sequencing, impulsivity, visuospatial, memory and/or loss of consciousness or control.

Intuitively, this list is expansive enough to range over various cognitive impairments like dementia and Alzheimer’s Disease, even if they are not singled out for special or explicit attention.

See: “What does Oregon State Law say about Driving with Alzheimer’s?” which is a subsection of my longer, and more comprehensive (but certainly not exhaustive) reference, elsewhere on this website.

Pennsylvania

According to Schemery Zicolello Law Firm, the Pennsylvania Medical-Reporting Law requires health care personnel to report to the Pennsylvania Department of Transportation the full name, date of birth and address of every person over 15 years of age that is diagnosed as having a disorder or disability that could impair his/her ability to drive.  In the list of reportable medical conditions, the attorneys specifically call out dementia.

That’s good enough for me.

If you want a bit more detail, scroll down to “Pennsylvania,” on my article, HERE.

Utah

Utah was mentioned in an article by National Center for Biotechnology Information (NCBI) as a reporting state. However, in the Fact Sheet for Patients and Families provided for Utah drivers, the indication for mandatory reporting by physicians is not present.

The document does state that the patient is expected to report and disclose their impairments to the department of motor vehicles.

So… I don’t find that Utah is actually a mandatory-reporting state.

For more, see: https://alzheimersproof.com/u-s-laws-on-driving-with-mci-and-alzheimers-disease/.

Concluding Remarks

Bear in mind that just because a state does not have mandatory-reporting laws, it should not be assumed that physicians won’t report anyway.

In the first place, a doctor might believe that it is his or her ethical duty to report a potentially dangerous driver – regardless of whether state law strictly requires that he or she do so.

Additionally, the physician’s other professional entanglements – for instance, his or her affiliation with a medical group or his or her professional-liability insurance – might have stricter requirements governing action steps in relevant cases.

Additional Information Or Resources

Over the course of several months, I compiled an article as a reference to U.S. Laws on Driving with MCI and Alzheimer’s Disease which provides a guide on all 50 states and their associated laws.

Another of my references is the post Canadian Laws on Driving with MCI and Alzheimer’s Disease which outlines comparable laws specified within the Canadian provinces.

You might also want to consult the National Highway and Traffic Safety Administration at their website: https://www.nhtsa.gov.

Moreover, in the Physician’s Guide to Assessing and Counseling Older Drivers, Chapter 7, Legal and Ethical Responsibilities of the Physician, some important key terms are defined.  Here are a few highlights.

Mandatory Medical Reporting Law States:  In some states, physicians are required to report patients who have specific medical conditions (e.g., epilepsy, dementia) to their state Department of Motor Vehicles (DMV).  These states generally provide specific guidelines and forms that can be obtained through the DMV.

Physician Reporting Law States: Other states require physicians to report ‘unsafe’ drivers to their state DMV, with varying guidelines for defining ‘unsafe.’  The physician may need to provide (a) the patient’s diagnosis and (b) any evidence of a functional impairment that can affect driving (e.g. Results of neurological testing) to prove that the patient is an unsafe driver.

Physician Liability States:  Case law illustrates situations in which the physician was held liable for civil damages caused by his/her patient’s car crash when there was a clear failure to report an at-risk driver to the DMV prior to the incident.

On the NHTSA’s website, a survey of medical review practices can be found that indicate while all 51 jurisdictions accept reports of potentially unsafe drivers from physicians, only 6 jurisdictions require physicians to report drivers to the motor vehicle agency.

Another helpful article on NHTSA’s website is ‘Current Screening and Assessment Practices’.  This article summarizes the legal requirements in each of the six mandatory reporting states.

Another helpful resource is the National Center for Biotechnology Information, the website for which can be found, here: https://www.ncbi.nlm.nih.gov.

National Library of Medicine, National Institutes of Health contains an article within the Journal of General Internal Medicine (abbreviated “J Gen Intern Med”).  Of interest is an article titled “Reporting by Physicians of Impaired Drivers and Potentially Impaired Drivers,” which mentions that virtually all states have established policies for the identification of drivers with physical or mental impairments, however, the reporting is voluntary with the exception of specific states.[1]

Additional references on AlzheimersProof.com include:

Is it Legal to Drive with Alzheimer’s Disease?

This is a resource regarding the legalities of driving with Alzheimer’s or another cognitive disorder.  This is not legal advice, simply research and research leads presented in a comprehensive article to assist in locating additional sources for further information. You can find additional information and important aspects specific to your state within my 50-State legal guide, HERE.

Are Drivers with Alzheimer’s more Dangerous than Others?

The above article, also on my website, goes into a discussion about a number of factors that may impact a person’s ability to drive, given their cognitive or mental state. This not only addresses that Alzheimer’s Disease occurs in stages (for more on which, see HERE), but also addresses some concerns around other cognitive impairments.

Is it Safe to Drive with Alzheimer’s?

I also have an article – and a companion video (see my YouTube channel, HERE) – talking specifically about considerations of driving safety, apart from an explicit discussion on any legal requirements.

Disabling Devices – Car

Now, if it gets to the point where you have to actually restrict your loved one’s access to the vehicle (and I sympathize with you if this happens), then you might find something of use in the above resource.

How do you Alzheimer’s Proof a Car?

The final piece that I’ll note, here, is a general article that I did on the question of how to “Alzheimer’s Proof” a car. Recall that Alzheimer’s Proofing is akin, in this context, to baby proofing or childproofing. For more on this, see HERE.

Disclaimer

Please note – this article should not be treated as providing legal or medical advice, but purely as a reference that provides general information in regards to the laws that require a physician contact a licensing bureau in regards to a patient’s ability to operate a vehicle. This post is given as-is; I do not warrant that the information is accurate or complete. But I provide the information in good faith and I believe, to the best of my knowledge, that it is reliable.

Notes:

[1] As noted in the main text, the article’s list of states includes Utah, while in other references, Utah is not mentioned as a mandatory state.

Warning Signs of an Unsafe Driver: Alzheimer’s Dementia

Alzheimer’s Disease is a progressive, neuro-degenerative illness that presents itself in stages. While there is some debate over the precise number of stages (for more on which, see my ARTICLE or companion VIDEO), for present purposes, we can say that there are three: early, middle and late. Because cognitive degradation is just what it is to have some form of dementia, the fact is, at some point, an Alzheimer’s sufferer will become unsafe behind the wheel of a car.

In the early stage of Alzheimer’s, memory impairments may be mild affording a person the ability to safely drive. A larger concern occurs during the transition from the early to middle stage of the illness.

During the middle stage of Alzheimer’s, memory impairments may become more severe. You or your loved one may be doing fine one day, but have a lapse in memory or judgement, causing challenges the next day. These lapses, or deterioration in memory, can make a person with Alzheimer’s more of a danger to themselves or others, especially when driving.

By the time a person reaches the late stage of Alzheimer’s, he or she will have lost so many abilities (including language, mobility, responsiveness, etc.) that driving will be virtually inconceivable. Generally, by that time, the level of physical disability alone makes operating a motor vehicle practically impossible.

But before that threshold is reached, it may be somewhat difficult to identify the precise time at which a person becomes an unsafe (or at least unreliable) driver. Here is a breakdown of some early signs or symptoms that may indicate it is no longer safe for a person with this illness to operate a vehicle.

(These signs may be noticed by the person themselves, a loved one, vehicle caretaker or an outside party such as a neighbor, physician, police officer, etc. Additionally, it is important to assess behaviors and signs relative to a person’s “baseline.” In this context, a baseline is the starting point for comparison. It’s basically the way a person acted or the degree of functionality they possessed for most of their adult life – at a point when they were obviously not impaired in the relevant sense.[1])

Emotional Changes

A person with a mild demeanor may become more aggressive, hostile, or otherwise difficult. These changes in behavior could potentially be due to a mood disorder or other neurological problem. However, these behavioral changes can also occur in a person with Alzheimer’s Disease or some other form of dementia.

If you notice that you or a loved one becomes unable to control anger or depression, becoming so overcome with emotional fits or episodes, it may be an indicator of Alzheimer’s or some other serious condition. Whatever the cause, these emotional changes will increase the dangers and risks of being behind the wheel of a car.

  • Anger – Your loved one may become agitated or irate with little or no apparent provocation, or beyond what a normally functioning person would think of as justified or reasonable.
  • Anticipation – In this case, the pathological component may be another outgrowth of memory loss. If you loved one seems or professes to expect or hope for an imagined or past event, it may be a sign of worsening dementia.
  • Disgust – An Alzheimer’s patient may have difficulty masking emotions and might display naked aversion, distaste, loathing, revulsion, etc. even in inappropriate contexts or directed toward things that they previously felt fondly toward.
  • Fear – On a low level, this might result in an increasing level of anxiety. It could be apparent through spoken language, but it may only become apparent to people who notice subtler signs or body language (e.g., in facial expression or posture) or habit (such as hand wringing or nail biting). Alternatively, fear may manifest in more extreme ways, for instance, through indications of a pathologically paranoid or suspicious turn of mind.
  • Joy – We all like to see our loved ones content and happy. But, in some cases, the level of jubilation or excitement may exceed what is appropriate to or warranted for the occasion. For instance, your loved one may not exercise due restraint. Alternatively, it may appear strike you as inappropriate in the sense that he or she seems to be taking pleasure in something that ought to elicit a different emotional reaction.
  • Sadness – A dementia-afflicted people might suffer from apathy or depression, or from some other related sort of unrelenting melancholy. They may tend to brood or become removed or withdrawn from family or other social circles.
  • Surprise – Look for evidence or amazement or bewilderment that seem out of place. Astonishment or shock could be a result of affected judgment or reasoning. But it also could be a function of increasing forgetfulness and impaired memory.
  • Trust – This may be related to fear and paranoia in the sense that your loved one may begin to suspect family members of plotting against them. Contrariwise, it might be expressed in an inappropriate or unwise willingness to follow or receive instructions from strangers.

Memory Changes

Some Alzheimer’s-afflicted individuals may have issues with short-term memory loss but are able to remember things that occurred further back in time. Vehicle operations can be broken up into “before,” “during” and “after” categories to determine if memory changes impact or undercut safe driving. Here’s what I mean.

Before Getting Into the Vehicle, Ask…

  • Do you or your loved one have an issue finding the keys?
  • Is it consistently a challenge to remember where the vehicle is parked? Or…
  • Is it hard to recall which vehicle belongs to you?

Everyone misplaces things from time to time. Obviously, when it is sporadic, this is not necessarily an indicator of Alzheimer’s Disease or some other form of cognitive impairment or dementia. However, the issue occurs when this becomes more of a pathological condition where memory loss is more frequent than normal, or when its occurrence undermines safety.

While Inside of the Vehicle, Ask…

  • Do you or your loved one have trouble remembering which pedal is the accelerator and which is the brake?
  • Is there confusion on what traffic signs or signals are indicating?
  • Do you or your loved one regularly forget where you are going, or how to get there?
  • Beyond the perceptual diminishment that often accompanies normal aging, cognitive impairments may erode sensory faculties. Does the driver have reduced sensory-processing capabilities? (See further below.)
  • Do the A/C or radio controls confuse you or distract you from the road?

A person with this illness may not understand signs to where they inadvertently drive through a construction zone. They may also forget where they are going or how to get where they are going, even if the route is familiar.

After Exiting the Vehicle, Ask…

  • Are there dents or scratches that cannot be accounted for?
  • Is parking haphazard or erratic?
  • Are items forgotten inside the car inadvertently?
  • Are keys routinely left in the vehicle?
  • Is the car left running unintentionally?
  • Have you or your loved one begun to receive mailed traffic citations? Or…
  • Is the driver being pulled over more frequently than before?

A person afflicted with this some form of dementia or other cognitive difficulty may not remember how a particular dent or scrape appeared on the vehicle.

All these items are warning signs or indicators that something else may be going on beyond normal forgetfulness.

Motor-Skill Changes

Cognitive disabilities are only half of the picture, though. Driving also places physical demands on a person. Therefore, various physical impairments can also undermine safe-driving abilities.

Similarly to the previous category, these changes can also be evaluated by looking at requirements that impose themselves before, during, and after using a vehicle.

Before Getting Into the Vehicle, for Example…

  • Changes in motor skills or reductions in physical strength may hinder a person from doing something as simple as opening or closing a vehicle door.
  • You or your love done may struggle with opening or closing the hatch or trunk.
  • It may become difficult or practically impossible properly open or close the hood of a vehicle.

It is true that memory may come into play here as well. And there’s little question but that forgetting how to do these minor tasks may be an indicator of Alzheimer’s.

But diminished motor skills can be serious problems on their own. And, when determining a person’s driving fitness, physical abilities need to be assessed alongside cognitive ones.

While Inside of the Vehicle, for Example…

  • A person with diminished motor abilities may have difficulty turning the wheel.
  • He or she may have trouble physically depressing the pedals or shifting gears.
  • Someone might find it physically taxing to keep the car in the correct lane.
  • Parking the vehicle may begin challenging, especially if the individual has limited abilities to turn their body or head.
  • Sensory impairments such as hearing or vision loss can also undercut the ability to safely operate a car or truck.

After Exiting the Vehicle, for Example…

  • Do you or your loved one have difficulties just physically getting in and out of the house, opening entry or garage doors, carrying groceries, and so on?

Of course, people often drive in order to assist them with the functional activities of life – such as shopping. While general impairments may not specifically speak to the question of safety behind the wheel, they do speak to the issue of whether driving is beneficial or necessary. If you or your loved one can no longer perform or engage in other life activities – like shopping – then it may be that driving is no longer helpful. This suggests that it may pose a risk that outweighs any potential benefit.

Additionally, the loss of non-driving-specific abilities indicates a general physical decline any may suggest either that driving-related abilities may soon erode or that further cognitive impairment is on the horizon.[2]

Clearly, being unable to physically control a vehicle, while not an Alzheimer’s-specific problem, may be an indicator that you or your loved one can no longer reliably or safely operate a vehicle.

Perceptual Difficulties

The normal aging process can cause perceptual difficulties. However, Alzheimer’s can exacerbate these issues.

Perceptual difficulties may impact visual-spatial abilities such as being able to judge distance or peripheral vision. Being spooked by shadows or not noticing cars or pedestrians next to you may be a result of diminished perception.

There may also be an inability to see traffic signs, signals, or other drivers (for example, people braking in front of them or merging into the same lane).

Perceptual difficulties may also include a decreasing ability to hear sirens or horns. This poses an obvious danger – especially, though not exclusively, if it is coupled with memory impairments. Forgetting what emergency-vehicle sounds indicate or require in terms of courses of action can lead to accident, injury, or death.

Reasoning Changes

A deviation from a normal route may cause confusion for a person with Alzheimer’s, as they may not be able to react or think through an alternate route. They may also have issues navigating around accidents or construction sites.

A person with this disease, or some other form of cognitive impairment or dementia, may have a reduced reaction time that, in turn, undermines their ability to react to unexpected changes within their route. These include such things as closed roads and bridges, etc., which could potentially lead to accident, and certainly engender confusion.

Deviations and detours can also result in you or your loved one becoming lost.

Concluding Remarks

As difficult as this can be for the afflicted individual, there will come a point where having them behind the wheel of a car or truck will become more of a danger than a benefit.

For more information, please see my companion YouTube video, HERE.

Postscript

In addition to these safety considerations, there may also be legal implications and potential liabilities in regards to a person driving with Alzheimer’s Disease.

While I am not a lawyer or legal expert, I do have several, relevant resources available on AlzheimersProof.com.

For instance, I have:

Additional Resources

Notes:

[1] Of course, some persons may have lifelong afflictions or disabilities. These will have to be factored in as well. Doing so is beyond the scope of this article. Consult a healthcare or medical professional for personal evaluations or recommendations.

[2] See, e.g., Salynn Boyles, “1st Signs of Dementia May Be Physical,” WebMD, May 22, 2006, <https://www.webmd.com/alzheimers/news/20060522/1st-signs-of-dementia-may-be-physical>.

Reduce Elopement/Wandering: ‘Defender’ Lock for Alzheimer’s

This one simple device may reduce 'elopement' risk

One of the major challenges in caring for someone who has Alzheimer’s Disease, or some other form of dementia, is that they are often prone to “elopement.” Of course, in the context of long-term care, elopement has nothing to do with illicit or surreptitious marriage. It has to do with a cognitively impaired person leaving the safety of the care environment without supervision.

This problem can be extremely vexing for the caregiver and perilous for the Alzheimer’s sufferer. I know this firsthand. On one memorable occasion, my dad was returned to his home by a police officer after he had been found wandering along a busy road in our city. During a snowstorm.

However, I was able to hinder my dad from wandering with an ingenious little gadget. In this article, I’m going to explain how to install that device – a door blocker called the “Defender.”[1] I have installed several of these blockers: one in an apartment, and a couple of them in a townhouse. I absolutely love these products. And I think that you might, too.

Introduction

What’s the Function of the ‘Defender’?

Knowing your loved one can open doors and leave the care environment without you or a caretaker in tow – or even realizing your charge is gone – is a terrifying proposition. Memory and reasoning deficits raise the possibility that he or she could become disoriented and lost, or even injured or worse.

From the perspective of long-term home care, then, the main benefit of these locks is to prevent your loved one from eloping. The devices are inexpensive, and the contemplated modification may be performed relatively easily.

This sort of change is part of what I mean by “Alzheimer’s proofing” your home environment. (This is a concept that I describe more fully HERE.) Under this way of using the phrase, it is a close cousin to childproofing a house as expectant parents would do in preparation for the arrival of a newborn.

Devices Double as Theft Deterrents

These door blockers can deter Alzheimer’s sufferers, or other cognitively disabled persons, from opening exit doors, leaving the care environment, and potentially putting themselves in harm’s way.

But it is worth noting that this particular use – keeping someone inside the house – is a bit of a departure from the product’s stated purpose.

This style of door lock has been designed and marketed as a security device and theft deterrent. When properly installed on an entrance door, the lock helps to prevent forcible entry into a house. It essentially fortifies the door so that even if a thief has jimmied it, or defeated its deadbolt and locking mechanisms, the door blocker will enable the door to remain in a closed position.

So, beyond the standpoint of Alzheimer’s proofing, it will add another layer of security to your home.

Can Your Alzheimer’s-Afflicted Loved One Defeat the Blocker?

As stated, the Defender is intended to be set into a locked position inside the home in order to reduce the probability of forcible entry should an intruder attempt to gain access to the interior of your house.

Given this, it has been designed to be locked and unlocked by normal-functioning adults. Therefore, it is possible for a cognitively impaired person to operate the latch. However, to do so he or she would have to have a number of cognitive and physical abilities. A rough-and-ready enumeration of these might look the following.

Cognitive Abilities Plausibly Needed to Defeat the Blocker:

  • The ability to notice the device
  • The capability to identify its function
  • The capacity to understand or the resourcefulness to determine the correct way to unlock it once
  • The motor skill and dexterity required to execute the unlocking action
  • The memory power to remember how to defeat it on subsequent encounters

I’m certainly no medical or psychological expert. But, the likelihood that a cognitively impaired individual would possess this collection of abilities seems to me to be somewhat low. More guardedly, I suppose that I would venture the opinion that the probability is low at least once your loved is sufficiently advanced in his or her dementia to be a serious elopement risk.

Often, if it is positioned high enough on the door – or if it is camouflaged – your loved one may not even recognize that the door blocker has been installed. Of course, this is because Alzheimer’s tends to diminish perception. In the case of my dad, for instance, I don’t believe he noticed that the thing was even there.

Additionally, a high-up position might hinder the door-opening ability of a senior adult in general – quite apart from any dementia – for example if he or she has joint or mobility issues.

Moreover, the door blocker requires an additional motion, outside of those used to open doors in the usual way, that may frustrate a person with a cognitive deficit and prevent them from eloping from the home.

WARNING!

I should, however, inject an important word of caution.

These door blockers may hinder your and your loved one’s abilities to exit the house in the event of a fire or other emergency. If you’re going to use these products as a deterrent to help prevent elopement of a loved one from the residence, you may want to ensure the blocker is activated only during the time periods where your loved one is at the highest risk of vacating the premises without you noticing.

Of course, from a theft-deterrence perspective, it is appealing to have the latch activated throughout the night or during whatever intervals occupants of the house wish to lower the likelihood of intrusion.[2]

Disclaimer:

I cannot advise you as to the appropriateness of any particular course of action for your application. Furthermore, I cannot be sure, and do not warrant, that the device will be effective for you – for any purpose whatsoever. This information is presented as-is, for general or entertainment purposes only. Whatever use you put this information to is entirely your own responsibility. No one at or affiliated or associated with AlzheimersProof.com assumes any liability for how you may implement or not implement any of the ideas described in on this website or in any companion videos.

Be aware of where and under what circumstances you are installing these blockers. The upshot is that I am certainly not guaranteeing that these devices or installation methods will be successful or safe for your individual or family situation.

I can only state that these blockers were a tremendous help for me and my family when caring for my Alzheimer’s-afflicted dad.

My Installation Procedure

‘Unboxing’ & Collecting Required Tools

'Defender' Install Sheet
‘Defender’ Installation Instructions

Upon opening the Defender’s packaging, you will find several items. These include:

  • The door blocker device itself;
  • Instructions;
  • 3 large wood screws;
  • 4 smaller metal screws;

Tools

The main tools needed include:

  • A drill (this could be theoretically be corded or cordless; mine was the latter)
  • 1/8-inch drill bit
  • Philip’s screwdriver

Optional (& Possibly Necessary) Tools

  • Wood chisel (if there is insufficient space in between the door and door jamb to accommodate the blocker)
  • Hammer (to use the chisel, if one is needed)
  • Center Punch (to tap a small “pilot” for the drill bit)
  • Tape Measure (to assist in positioning the blocker on the door with respect to the other locking hardware – such as doorknobs and deadbolts)

Safety Equipment

  • Safety goggles

Positioning the Blocker

You may want to begin by determining where you want to place the Defender door blocker. The instructions suggest that it be installed at least 6 inches above the door handle and deadbolt.

As suggested above, it may be beneficial to install this blocker a bit higher than this. In fact, there is an “L-shaped” area on the door within which the manufacturer recommends placing the Defender.

'L'-Shaped Area
Inverted ‘L’-Shaped Install Location

While determining where to install the blocker, place the blocker in the locked position. While in this fully closed position, slide the plate between the door frame and door with the black pad against the door.

This will allow you to get a good look at everything, and to reposition the blocker is desired or necessary.

**Be mindful of any electrical outlets nearby as there may be wiring within the walls near where you may be drilling. Additionally, look at surrounding door hardware, molding, key hooks, or other things that might interfere with your ability to operate the blocker and allow it to swing fully from locked to unlocked positions, and back again.

Actual Installation Steps

Step One

Once you have determined where you want to place the door blocker, use a pencil to mark the placement of the center screw within the door frame.

Step Two

Use your drill and 1/8-inch drill bit to make a hole where the center screw will be placed.

Step Three

Place the door blocker plate back against the door frame, allowing the recently drilled hole drilled to be visible in the center-hole of the plate.

Step Four

Drive the center screw into the door frame allowing the blocker plate to be provisionally positioned.  The center hole is oval shaped and allows for the back-and-forth movement of the device in order to ensure that it is tight enough against the door to perform its function, but not so tight that it prevents you from latching and unlatching it.

Step Five

Test for the appropriate and desired fit. Before fully securing the door blocker, close the door and test the blocker in the locked position to ensure the placement is optimal. If the lock is difficult to engage or disengage, the placement of the plate may need to be adjusted slightly for proper operation of the device. In this case, you would simply loosen (but not remove) the center screw, move the blocker, and re-tighten the screw.

Step Six

Once the positioning has been established, ensure that the center screw is fully tightened – but not over-torqued. (You don’t want to strip the head or threads.)

Screw assortment
Longer Wood Screws & Shorter Metal Screws

Then, drive in the remaining three (3) screws – large screws for a wooden door frame; small screws for metal – into the remaining holes present on the door blocker plate to firmly secure the blocker.

Congratulations!

Your Defender door blocker has now been successfully installed!

Open & Closed
‘Defender’ in Position

If you need an additional assist, or a bit more in the way of visual aids, never fear. For a demonstration of the operation of the lock, or for a video tutorial on the installation procedure, please view my companion YouTube video: Alzheimer’s Elopement & Access Control: Install the Defender Door Lock.

Thank you for reading! I wish you all the best trying to deal with elopement risk.

Notes:

[1] The “Defender” appears to me to be an off-brand version of a different door blocker known as the “Door Guardian.” I also have the Door Guardian and will walk through its – nearly identical – installation procedure in a subsequent post.

[2] Note that there is no “key” and that the device is not designed to be unlocked from the outside. It is supposed to be activated and deactivated from within the area being secured.

Custodial Vs. Skilled Care: What’s the Difference? Who Pays?

Different Types of Care: Custodial & Skilled

“Custodial care” is nonmedical help with the Activities of Daily Living (like bathing and eating). “Skilled care” is medical care, such as giving medications and shots, dressing wounds, drawing blood, and so on.

The phrase “long-term care” can be confusing, since custodial care is sometimes used as a synonym for custodial care, and sometimes it’s used for the combination of custodial and skilled care that a person might receive in a nursing home.

I would like to spell these things out as simply as I can.

So, let’s first think, on a very basic level, about what we mean by long-term care.

A Personal Example: My Dad

As I have written elsewhere, my dad, Jim, passed from Alzheimer’s disease in 2016. I took care of him in his own home for four years. And then he was in a nursing home for four years after that.

That means that he suffered obviously from Alzheimer’s for at least eight years. In fact, it was longer. We can reasonably conclude this for two reasons.

Firstly, I started caring for him was because it was becoming increasingly apparent that he needed help. So, we are justified in believing that he was suffering from some cognitive impairment before I assumed the role of his daytime caregiver.

Secondly, scientists inform us that the brain degeneration of Alzheimer’s begins before any symptoms manifest themselves. Therefore, we can infer that Jim had the very beginnings of Alzheimer’s prior to anyone noticing that something was wrong with him.

But, once we became aware, we observed that he needed help with the basic tasks of everyday life.

Help With Everyday Activities

Called “Activities of Daily Living,” or “ADLs,” these include things like bathing, dressing, eating, maintaining continence, toileting by yourself, and transferring in and out of bed.

These are things we all need to do each day. That’s 24/7/365, as it were.

That’s the intuitive definition of “long-term care.” It’s help with the ADLs, delivered over a fairly long period of time.

So, what’s in view here is a person who has a chronic condition, disease, disability, or whatever and who is going to require everyday care over a long period of time.

Probably Also Need Extended Medical Care

It turns out that many conditions that will prompt this level of care are also terminal conditions. (I go into greater depth on terminal illnesses HERE and HERE. And I have a video on the topic, HERE.)

A personal who is so severely incapacitated that he or she needs daily assistance with the basic activities of living most likely also requires some kind of ongoing medical care.

Think about my dad, again. He had Alzheimer’s Disease. He required both medical care and nonmedical care. He needed supervision and medication, for instance.

As noted previously, one way of using the term “custodial care” is applying it to nonmedical care. But, a person – like my dad – who has a chronic and debilitating illness may very well also require medical care (e.g., pharmaceutical interventions, speech therapies, and so on). When we say “skilled care,” then, we’re thinking of this latter sort of care.

Possible Word Confusion: ‘Long-Term Care’

Once again, the intuitive difference between custodial care and skilled care is going to be that custodial care is essentially nonmedical / supervisory care, whereas skilled care is going to be medical.

Now there are a few different ways of kind of getting more clear on what the differences are, and I’m going to talk about what the actual care is in terms of (1) what is involved, (2) who provides the care, (3) where the care is provided, and (4) how you pay for it.

But before I do that, let me just say one another word about long-term care.

Sometimes, often in everyday speech, the word “long-term care” is used to encompass both custodial and skilled care. In this loose sense, long-term care basically means “whatever your loved needs over an extended period of time.”

A person may think about a relative who’s in a nursing home – like my dad was. My dad received both types of care in the nursing home. So, it’s tempting to think that my dad’s long-term care had medical and nonmedical elements. He needed both – over the long term.

Other times, the word “long-term care” is essentially used as a synonym for custodial care. This use is common in billing applications, healthcare insurance, long-term-care insurance, Medicare, Medicaid, etc.

The upshot is: understand the context of the word use. And, when you hear it, be sure that you know which definition of the word is operative!

‘Qualifying’ for Long-Term Care

What I mean, here, is essentially this: What are the diagnostic triggers that would prompt a doctor to say, “This person needs long-term care”?

This is an important consideration, especially – but not exclusively – for people who may have private nursing-home and other pertinent insurance policies. Long-term-care insurance generally “kick in” once the insured person is certified as being in need of long-term care.

2 Triggers

Physical

This is measured in terms of the “Activities of Daily Living,” or ADLs. In most materials, you’ll find six of these listed. I mentioned them, above: Bathing, dressing, eating, transferring, toileting, and controlling bodily functions.

From the physical point of view, to be “long-term-care certified” essentially means that you lack two out of six of these activities. So, if you’re incontinent and unable to feed yourself, then that would count. Or, if you’re unable to dress yourself and get in and out of bed by yourself, that would count also.

Your needs will have to be evaluated and documented by a licensed healthcare professional, chiefly, a physician.

Cognitive

But there’s also a cognitive or mental trigger.

You may also be certified as in need of long-term care if you have a cognitive impairment to a significant enough degree that you would require more or less constant supervision in order not to hurt yourself or other people.

Sometimes this trigger is pulled at the same time as the physical one, and other times the two are quite separate. In my dad’s case, for instance, he was physically able-bodied (in terms of the ADLs). He had no physical difficulties.

His problem, initially, was purely cognitive. Of course, as the disease progressed, his condition worsened and then he met the long-term-care qualification “tests” multiple times over. This is not uncommon.

Lacking Activities of Daily Living = Custodial Care

When you think of these Activities of Daily Living – bathing by yourself, dressing, feeding, and so on – think custodial care. Likewise, think of custodial care when you think about severe cognitive impairment that necessitates supervision.

Custodial care is going to be that kind of care that assists a person in receiving and in performing the Activities of Daily Living or providing the needed supervision.

Medicines, Medical Tests, Therapies, Etc. = Skilled Care

But my dad’s Alzheimer’s Disease also required certain pharmaceutical interventions. He was on put on the drug Aricept, for example. And it had to be administered to him at various times.

Of course, family members are able to administer that kind of medication at home. But in an institutional care situation, you wouldn’t want a person who doesn’t have proper licensing and proper credentials to administer drugs to your loved one.

But, as of this writing, people don’t need to be licensed to help bathe your loved one, or to help feed him or her.

Therefore, a second distinguishing feature of custodial care is that the care practitioners do not need to be licensed.

On the other hand, skilled care, as the medical portion of care, does require special training and licensing. In fact, that is one of the reasons it is referred to as skilled care.

Nursing Homes Provide Custodial & Skilled Care

In certain care environments, you are going to see a combination of these needs being met. So, for instance, a nursing home is a place that is going to provide both custodial and medical care for its residents. It’s a one-stop shop, so to speak.

One of the reasons why these definitions get a bit mixed up is because of the fact that we see our loved ones in this kind of composite care environment. It’s therefore natural to assume that everything going on in that environment is appropriately called “long-term care.”

Possible Care Environments

Of course, other care environments exist beside the nursing home.

Home Care

Number one, a person could receive care in his or her own home, or in the home of a friend or relative. Certainly, you can have people come into the home to provide the kinds of custodial supports that a person might need. And, generally speaking, if you’re receiving care in the home the professional care is probably going to be a supplement to a familial caregiver.

When my dad was living in the home, it was my mom and who were giving the supports that were necessary for him to get through the day. But, if we needed additional help, then we might call somebody else to the house.

This extra support could be skilled – like a visiting nursing – or unskilled/custodial. But, for the most part, we handled the custodial portion of the care ourselves.

Assisted Living

An assisted-living facility is a place that’s going to help people who are starting to have difficulty with the Activities of Daily Living. Such facilities try to emphasize and encourage independence. Seen from a different perspective, these facilities actually require a fair degree of independence – since they are generally not licensed or staffed to provide the level of care available in nursing homes.[1]

Nursing Home

But, for present purposes, the third main care environment is a nursing home.[2]

A nursing home is going to provide a level of care that’s a notch above an assisted-living facility in terms of comprehensiveness.

Whereas an assisted-living facility is equipped to provide low-level help with some daily activities, a nursing home is able to provide full-blown custodial care.

And whereas an assisted-living facility likely has a nurse or doctor on call, a nursing home always has medical practitioners on the premises. Skilled care is part of the overall care provided by the home.

To put it slightly differently, nursing homes provide comprehensive care services.

Billing of Services

Why are these distinctions so important?

Well, the distinctions become important largely when it comes time to pay the bill!

As usual, this discussion can become nuanced and detailed. But here’s the bottom line.

There are three main ways to pay for long-term care services, broadly construed.

Private Pay

“Private paying” is where you pay for yourself. You’ll likely do this out of your own assets (e.g., checking, money market, savings accounts; emergency funds; retirement vehicles; etc.) or your own income (for instance, annuities, pensions, rental payments, social security, and so on).

While you pay for yourself, you can go wherever you can afford. You can have whatever accommodations and services you like. At least, you can have all this so long as your money holds out.

At the level of nursing-home care, your bill is likely going to be a composite of skilled nursing services and custodial services. But if you’re paying entirely out of pocket for your care, then it doesn’t matter which sort you’re being billed for. You pay for everything yourself, regardless of what type of care it is.

So, in the private-pay scenario, the custodial/skilled distinction really doesn’t matter from the standpoint of your checkbook.

Government Aid

Medicaid

Once you “spend down” your assets to a particular – and low – level, you may be eligible for Medicaid. This is a government-assistance program that is geared towards helping impoverished people pay for necessary medical expenses.

For qualifying individuals, Medicaid pays for (portions of) both custodial and skilled care. However, there are strict asset and income tests that are applied.

Moreover, your preferred facility may not be able to accommodate you. Some facilities have no or few “Medicaid beds.” Others require that you have lived at the facility under private-pay arrangements for a specified period (not infrequently one or more years) before you can claim a Medicaid bed. Others have long waiting lists.

Typically, the relevant beds are in semi-private rooms with at least one other occupant. Additionally, Medicaid usually segregates its recipients by sex.

Medicare

What about Medicare?

For purposes of this post, let’s just say that Medicare is a government program that provides healthcare coverage for seniors (i.e., people over the age of 65).

In principal, then, Medicare is available to cover at least some of the medical-related, skilled-care portion of the nursing-home bill. Now, there are several caveats.

One of these is that Medicare only really covers hospital bills and short-term skilled-nursing costs.

Additionally, there are requirements that the skilled nursing, to be covered, must follow a hospital stay.[3]

Curative Vs. Palliative Care

Finally, Medicare is geared toward what is called “curative care.” In basic terms, this sort of care is supposed to (help to) restore a person back to good health.

“Palliative care,” on the other hand, merely relieves symptoms – it doesn’t “cure” a patient.

You run headlong into trouble, here, when you start to consider conditions, like Alzheimer’s Disease, that have no cures.[4]

Elsewhere, I have discussed the question of whether Alzheimer’s is a “terminal illness.” (For a written article, see HERE. For a video touching the issue, see HERE.)

But suffice it to say that Medicare doesn’t generally cover palliative care – except in end-of-life, hospice situations.

The Upshot

The long and short of it is this, then.

Medicare only covers short-term skilled care and curative care.

Custodial and palliative care is generally not covered by Medicare at all.

Medicaid may cover these – is a person is eligible based on asset and income qualifications.

But that means that if a person is receiving both custodial and skilled care in a nursing home, and even if Medicare is paying for some of the medical/skilled portion of their bill, they cannot rely on Medicare to pay for any of the help they get with Activities of Daily Living. And they cannot hope to get Medicaid assistance until their own assets are spent down.

Long-Term-Care Insurance

Besides paying out of your own assets and spending your own assets down until you qualify for Medicaid, really the only other payment option is to use proceeds from a long-term-care insurance policy.

Such policies have a high application-decline rate, partly because people generally wait so long to try to purchase them.

Now I get more in depth into long-term care in other places. But, essentially, long-term-care insurance is the kind of insurance that’s going to help you to pay for the nonmedical portion of your bill. Long-term-care policies pay benefits when you need custodial care.

So, if you are getting help getting paying for the medical or skilled-care portion of your bill via your health insurance or via Medicare, then a long-term care insurance policy is able to pay for the custodial-care portion.

Of course, you have to be long-term-care eligible in order to collect payouts from this type of policy.

Basically, “eligibility” comes to this. You have to lack two of six of the aforementioned Activities of Daily Living, or you have to be cognitively impaired to such an extent that you require supervision.

But, once a physician “certifies” you as long-term-care eligible in one or both of these ways, then your long-term-care policy is supposed to pay out.

Only a few policies these days have lifetime benefits. So, you should be aware that is it possible to exhaust the proceeds of such a policy. But, it does constitute a third possible funding option, and can delay or avoid exhaustion of your personal assets.

Confusions Summarized

One confusion arises with respect to the fact that “long-term care” is a phrase that can be used for both types of care, together, or as a strict synonym for custodial care only.

A second confusion comes from the fact that both kinds of care – custodial and skilled – are delivered side-by-side, in the same environments. For example, and as discussed, you may get both medical and non-medical care in a nursing home.

Thirdly, there are confusions with respect to how care is billed. What you have to realize is that nursing-home bill is going to be a conglomeration of items, some of which are going to count as custodial services, and others of which are going to be skilled. Who pays – and what kind of insurance or assistance is relevant (e.g., health or long-term-care insurance, Medicare, or Medicaid) – is going to depend in part on this classification.

The Major Issue Restated

For most people, the main point of learning the distinction between custodial and skilled care is to gain clarify and insight into the question of how the relevant types of services can be paid for.

Who pays for a particular care service and for how long is, in part, a function of whether the service is classified as “custodial” or “skilled.”

Custodial services – like help bathing, dressing, and eating – can be provided by unlicensed and non-skilled care providers. These services may be delivered in a variety of care environments – from your own home, to adult daycares, to nursing homes – but, ultimately, these services are not covered by health insurance or Medicare.

So, you’re stuck paying for these privately (that is, out of your own pocket), through Medicaid (after you’ve exhausted your own assets), or through long-term-care insurance.

Skilled care – that is, the sort of care that is provided by licensed and skilled medical professionals – may be covered by health insurance and Medicare. But this is not always the case because of Medicare’s rules and because it only really covers “curative” interventions – as discussed, above.

Notes:

[1] In terms of whether or not a person would be safe to reside in an assisted living facility, or whether they require a higher level of care, sometimes people talk about various requirements. These are things such as being able to get to an exit by yourself, being able to get around without mobility aids, and so on. I’ll get into greater depth on these questions in subsequent posts or videos.

[2] There are also adult-daycare, hospice, and respite facilities. But I will get into those in another place. See my video presentation, HERE.

[3] Getting into detail about the precise rules, here, is beyond the scope of this article.

[4] Presently, at any rate.

Halloween Challenges, Dangers & Safety: Alzheimer’s Dementia

Halloween can be a fun time for young and old alike. For many, it’s their favorite time of the year. But, for the cognitively impaired, Halloween can present special challenges.

Caveat

Some of these tips are and have pros and cons.

There’s going to be a sense in which let’s say for example turning off the lights in order to dissuade trick-or-treaters might be an increased risk for tripping inside the home and it might be an invitation to vandalism or burglary. You may need to adapt some of these tips for your specific situation.

Two Main Concerns

Firstly, Halloween presents dangers for your Alzheimer’s-afflicted or dementia-stricken loved one. Secondly, people who are cognitively impaired may present challenges for others – not least, youthful trick or treaters.

Problems for Dementia Sufferers

Alzheimer’s Disease and other forms of cognitive impairment may result in anxiety, agitation, confusion, and numerous other emotional changes.

In general, these affective, cognitive, and physical difficulties are made worse during a holiday like Halloween.

One primary reason for this is that the normal trappings of Halloween can add to, magnify, or trigger negative states such as confusion and disorientation. It can exacerbate emotional problems like anxiety, agitation, and fear.

Additionally, Halloween can present certain physical dangers that go beyond what you would experience in the normal course of Alzheimer’s Disease.

Fire

There are burning hazards from candles, bonfires, jack-o-lanterns, and other things.

Choking

Depending on the stage of your loved one’s condition, you may have to contend with choking hazards from candy and other kinds of treats that are left out.

Falling and Tripping

Normal, age-related mobility issues are made worse by cognitive impairments. But the low-lighting conditions that often accompany Halloween can lead to increased risks of falls and trips.

Criminal Activity

In addition, and unfortunately, there are those people who are looking to take advantage of elderly people. People who are cognitively impaired (for example, who might be in early or middle stages), but who are still able to live alone part of the time, can be targeted and victimized in an array of ways.

On the severe end of the spectrum there are things like assaults and burglaries. On the less serious side, things like pranks, vandalism, and scams may escalate during holidays,

Cognitively impaired individuals may misperceive threats in a couple of different ways.

Number one, they may misperceive a person who is really an innocent trick-or-treater as a threat.

Number two, they might misperceive a person is actually a threat, as not being a threat.

Disorienting Factors

Doorbells and Knocking

Think about the amount of time the doorbell might ring during the evening. Or consider the effect that repeated knocks on the door might cause.

These can be startling at best. But they can actually be panic inducing at worst.

Of course, the actual effect will depend on the nature of your loved one’s affliction.

The geographical area and crime rate also play large roles, here.

Costumes and Masks

Another aspect of disorientation is the costumes and masks worn by party goers and revelers.

In the first place, some of these can be downright scary.

But, in the second place, they might make it even more difficult than usual for a cognitively impaired person to identify someone else – even if that person is (or should be) known or recognizable.

Alzheimer’s and other dementia-afflicted persons react poorly and even unpredictably to unfamiliar situations.

Masks and other kinds of costumes are going to obviously make it difficult to recognize a person. Predictably, this is going to increase the level of confusion that might be felt by some sufferers – or their caretakers.

Decorations

Halloween decorations can range from the grotesque and the off-putting to the downright terrifying. Be mindful of the effect these might have.

Horror Movies

Television, and other kinds of movies and “programming” can be horrifying and extremely scary, especially for young children and – again – those who are cognitively impaired.

Bear in mind that hallucinations and even paranoia are a couple of symptoms that typically go along with dementia. (For my article on this, see HERE.)

And these perceptual maladies are just a normal part of Alzheimer’s! Halloween just can make these kinds of things a little bit worse.

Cognitive and emotional difficulties are compounded by numerous factors.

Dim, Flickering, or Strobing Lights

There are often lighting changes.

So, for example, lights are often turned down low. This dim lighting might make it difficult for a dementia-affected person to perceive his or her surroundings or navigate the living environment.

This, in turn, can lead to falls and trips.

Some lighting might be flickering or strobing, which can also be disorienting. It might also cause seizures in individuals who are prone to epileptic-type fits.

Other changes might include candles and bonfires. These present numerous hazards.

Firstly, as previously mentioned, they may cause burns and house fires.

Secondly, they may give off smoke (and set off agitating smoke alarms) or even deadly carbon-monoxide gas. (For more on these dangers, and how to alleviate them, see HERE and HERE and HERE.)

Thirdly, they can also lead to an increased risk for tripping and falling inside the house. The flickering and uneven light can be just as disorienting as many novelty lamps or black lights.

Strange Sounds

Sounds might be loud.

Or they might be just plain eerie. And that can be unnerving.

Miscellaneous Dangers

Additionally, knives and other paraphernalia (for instance, those that may be used to carve pumpkins or set decorations) could be left lying around.

Dos and Don’ts

So, what can you do? (An abbreviated version of this list is online HERE.)

Don’ts

Hopefully it goes without saying, but don’t put your fun over your loved one’s well-being. Many people love Halloween and are ready to go all out. But remember, if you’re dealing with somebody who’s got a cognitive impairment. They may require special and a particular amount of love and care, especially during a time like this. You may have to sacrifice a few of your decorations and so on.

Don’t bring love ones to disorienting environments – out to malls (if there are any remaining!), to parties, or in general to places where there is bound to be lots of boisterousness or commotion.

Don’t leave Alzheimer’s suffers alone.

Don’t leave carving equipment lying about.

Don’t alter lighting beyond what would be safe.

This one is a bit tricky because, on the one hand, you’re going to want to leave lighting on inside the home for the purpose of helping your loved one to navigate the home environment. But, on the other hand, leaving the light on inside can signal to trick-or-treaters that you actually have a house that is worth visiting.

So, there can be some pressure to turn the lights down low in order to dissuade people from coming up to your door.

If you have good window treatments on the outside of the house or on the inside of the house, then you can turn the lights on inside without having to worry about that light penetrating to the outside.

If not, it might be better to retreat to an inner portion of the house where perhaps you can turn those lights on and leave the outermost lights off.

Don’t leave exterior or porch lights on, as it can signal the trick-or-treaters that yours is a house that you might that they might want to approach. (But, per my caveats, if you are in a burglary- or vandalism-prone area, then you may want to leave the lights on after all.)

Don’t over-decorate the house.

Don’t over stimulate your loved one. This can even include the opening and shutting of the doors.

If you are passing out candy, don’t overwhelm your afflicted relative with guests or parties inside the home (or outside of it, for that matter).

Dos

Do keep a careful watch over your loved one.

Do emphasize the lighthearted over the horrifying.

Do keep interiors well enough lit for safe passage.

Do remove the car from the driveway, if feasible. Placing it into the garage so that the house does not appear to be occupied.

HOWEVER… As I started off the video by saying, some of these tips have pros and cons. This is one such tip.

If you live in an area where you think that the house would be construed as vacant if it didn’t show signs of presence inside; or, relatedly, if you think that the lack of light or a vehicle might make the home more of a risk for a burglary attempt; then you obviously want to mitigate that risk by modifying or disregarding these sorts of tips.

So, again, you may have to adapt some of these tips for your own situation.

It’s also worth a discussion about whether your household should pass out candy or not. Obviously, if it’s going to disrupt the evening or if it’s going to disturb your Alzheimer’s-afflicted relative or loved one, then it’s probably not a good idea.

Some people advise placing candy outside, maybe in a bowl, and putting a sign on it that says, “Go ahead; take one; don’t ring the doorbell.”

Other people say, “No, you shouldn’t do that,” because candy left outside could be tainted or manipulated or stolen. Again, the sign could be interpreted as an indication that no one is home, which – depending on the area that you’re in – might be an invitation to criminals.

This is something you’ll have to figure out by yourself, given your situation.

Depending on your level of comfort, you might consider having someone else pass out candy for you. He or she could remain outside, provided that the weather is agreeable. If that person has whatever he or she needs, then this would reduce or eliminate the need to have to enter and exit the home very often. This would be a big help in terms of minimizing agitation. The question is: is it worth all that just to appease trick or treaters?

Do respond to signs of upset – whether this be due to overstimulation or agitation.

Some helpful tips are do set up some sort of a safe environment inside the house – a safe space, a room that’s especially quiet. You should have some quiet activities planned out and ready to go. These can be simple puzzles or photo albums. They could be familiar movies, music, and so on, or familiar books – something that you would be able to read to your afflicted relative.

Do reassure if necessary.

And do remove problematic decorations.

You might also consider just not being in the home (especially if you think you’re in a high traffic area). Different neighborhoods vary in this regard. Sometimes the neighborhood could be completely quiet and other times it might be extremely filled with activity.

Not to sound like a broken record but weigh the benefits of leaving against liabilities – for example if you think an unoccupied home would be a target. On the other hand, it might be a good idea to permanently relocate your loved one if they live in a high-crime area. Though, this is often easier said than done – especially if money is tight.

Disclaimers: First of all, there is no substitute for watchfulness any of the tips that I may provide are not supposed to be a substitution for diligently and vigilantly watching your loved one. Number two I am not a lawyer. I am not a doctor. I cannot give you specific advice I can give you specific Promises that any of the things that we will discuss will prevent injury and that is why watchfulness is so important.

Have a happy and safe Halloween!

Once again, for my quick-reference list, see:

https://alzheimersproof.com/halloween/

12 Mental Exercises for Staving Off Alzheimer’s (POSSIBLY)

Alzheimer’s is a degenerative brain disease that causes diminution of cognitive abilities, including memory, perception, and reasoning. As of this writing, Alzheimer’s Disease afflicts between 5.5 and 5.8 million people in the United States and between 44 and 47 million people in the world. It’s possible causes – discussed HERE – are not well understood. (There are widely mentioned RISK FACTORS.)

Various researchers, however, have suspected that at least some of the blame for Alzheimer’s can be placed on controllable things like diet/nutrition and exercise – both mental and physical. The general idea is that if you don’t “use it” (i.e., your brain), you might “lose it”![1] To that end, several sources have posited a slew of activities that are geared toward keeping you cerebrally fit. I’’ take a sort of “cocktail” or “grab-bag” approach.

Here is my list of the top twelve ways you might be able exercise your brain to prevent Alzheimer’s Disease. (See “Caveats,” below.)

Board and Card Games

An article in the British newspaper Independent related that “playing board games …could help” with mental decline – perhaps to an even greater extent than working crossword puzzles (about which, more in a moment).[2]

According to the results of one study that looked at brain scans: “Middle-aged people who [are] avid game players …[tend] to have bigger brains than people who [do] not play games…”.[3]

These more massive brains can confer a big advantage. Some people refer to this as “cognitive reserve.”[4]

Brain Teasers

“Brain teasers” are a type of game, usually consisting of problems, riddles, and the like of that that are solved usually for amusement. But what if they could serve a more useful purpose?

Numerous news outlets have reported on the possibility that various brain teasers, mathematics puzzles, and mysteries might help to enhance your cognitive health.

In the article “How to Outsmart Alzheimer’s,” Wall Street Journal columnist Amy Marcus reported that “quizzes and other cognitive challenges” might push back the onset of Alzheimer’s – “perhaps indefinitely.”[5]

So, reach for those puzzles and put your mind to work!

Chess

Chess is a two-player strategy game that has been around for hundreds of years. It’s played on a board composed of 64 squares of alternating colors. In total, there are 16 pieces per side (32 in all): eight pawns, 2 knights, 2 bishops, 2 rooks, 1 queen, and 1 king. Each type of piece has different rules governing its legal moves. The overall objective of the game is to “corner” (or “checkmate”) the opponent’s king in such a way as to leave it with no counterattack or means of escape.

Chess can be a very involved game with lots of subtlety and variety. It has competitive and social aspects (on the further benefit of which, see further on). But, on the other hand, it can be played over the internet without you (or your loved one) having to leave home.

Once again, some researchers suggest that “playing chess helps stave off the development of dementia.”[6] In fact, one study showed that playing chess “resulted in an almost 30% reduction in” dementia risk.[7]

Checkers

A two-player game, checkers is similar in some respects to the aforementioned chess. For instance, the board consists of 64 alternately colored – or “checkered” – squares.

Checkers is, however, played with 12 pieces per side instead of 16. Each piece is the same at the beginning of the game: simply a small, circular disk. The object of checkers is to “capture” or remove all (or at least most) of your opponent’s pieces or to leave him or her without any legal moves.

Although checkers has less variety in terms of pieces and moves, it is plenty rich in terms of move combinations and traps.

“Studies show games like checkers can boost your brain strength.”[8]

Crosswords

Admit it: Here’s the one you’ve probably been waiting for!

Simply put, a “crossword” is a kind of word puzzle. It is usually presented as a sort of grid with a combination of “empty” boxes and shaded boxes. The object of a crossword is to answer questions or use clues to fill in the empty boxes with words. Often, the words crisscross and interconnect in interesting ways – usually by sharing letters – which accounts for the name of this puzzle type.

Some investigations have suggested that working crosswords can boost mental ability and function.

Whether these activities affect age- or Alzheimer’s-related cognitive decline is an open question.

However, the National Center for Biotechnology Information, part of the National Institute of Health, published a study revealing that doing crossword puzzles delayed cognitive impairment – specifically, memory decline – by an average of two and a half years.[9]

Language

There’s a joke that goes something like this.

Question: What do you call a person who speaks three languages? Answer: Trilingual. Question: What do you call a person who speaks two languages? Answer: Bilingual. Question: What do you can a person who speaks only one language? Answer: American!

A quick Google search suggests that around 80-85% of Americans are monolingual.[10] Similar percentages apply in Canada. And the United States and Canada have some of the highest rates of Alzheimer’s Disease. For instance, it is the sixth leading cause of death in this country.

This is compared to approximately 45% of Europeans who are monolingual.[11]

Some research suggests that being bilingual can delay the onset of dementia.[12] For example, a 2013 article from CBS News is titled “Learning Another Language May Help Delay Dementia.”[13]

The article reported on a scientific study of various subpopulations in India. The suggestion was that speaking another language can push Alzheimer’s onset back an average of four to six years.

However, a key word is delay. Many people Belgium and Iceland are multilingual. However, both of those countries are in the top ten of nations with high percentages of Alzheimer’s dementia – according to WorldAtlas.com

In fact, Finland is the nation with the highest affliction rate. And a preponderance of the population appears to be bilingual to one degree or other.

Still, it seems reasonable to talk about a “protective effect of bilingualism.”[14]

Music

I have written a bit about how musical therapy can be a helpful intervention to explore when it comes to treating Alzheimer’s sufferers. (See my article “Can Music Calm an Alzheimer’s Patient?”)

A few studies have also led investigators to conclude that things like “playing musical instruments” can be better than working crossword puzzles or doing Sudoku. In fact, some suggest that this can “significantly reduce” a person’s risk.[15]

But for a more complete look at risk factors, see my video dedicated to that topic.

Puzzles

For those who weren’t introduced to these as children, jigsaw puzzles are basically jumbles of irregularly cut pieces (originally of wood, but now largely cardboard or plastic) that must be assembled in the correct order to reveal a pattern or picture. Pieces range in size from large (for small children or Alzheimer’s sufferers) to small (for people of normal to high cognitive function who may be looking for a challenge).

This deep into the article, you can probably predict what I’ll say next. “[J]igsaw puzzles …can help keep the mind active and a little sharper.”[16] (There are numerous kinds available. For my suggestions, see HERE.)

Reading

Some researchers believe that simply reading (books, magazines, etc.) frequently can have a protective and supportive effect on our brains. This could honestly be as mundane as picking up the daily newspaper. Or, for people who are more electronically inclined, visiting your favorite news website.[17]

If you walk to your local library, you could add a bit of exercise into the mix as well!

Social Interaction

According to a report from National Public Radio: “social interaction may be a better form of mental exercise than brain training,” where “brain training” refers to exercises designed to enhance processing speed and promote reasoning.[18]

Just “being around” other people can be of great benefit to Alzheimer’s sufferers.

Still, it is well to recall that causal direction is difficult to establish. Is it that social withdrawal leads to Alzheimer’s, or that Alzheimer’s leads to social withdrawal?

Sudoku

Here’s another – and more arithmetical – sort of puzzle: Sudoku. This Europe-originated puzzle with the Japanese name is essentially a reworked “magic square” in which numbers are inserted into a 9×9 grid. The object of the number game is to fill paper so that every column, row, and embedded 3×3 grid contains all numerals from 1 to 9.

One scientist stated: “…doing Sudoku isn’t probably going …to prevent you from developing Alzheimer’s disease” by itself.[19] Still, there’s little doubt from many investigators that “regular use of word and number puzzles” – like Sudoku – “helps keep our brains working better for longer.”[20] At least one scientific “study has identified a close relationship between frequency of number‐puzzle use and the quality of cognitive function in adults aged 50 to 93 years old.”[21]

If numbers are in your wheelhouse, give it a shot. If letters are more your thing, feel free to see our section on “crosswords,” above!

Working

You read that correctly. We’re talking about going to work.

Before you complain about your job, consider that, for many people, their job provides their “daily cognitive training.”[22]

This is to say that just going to work can have some neural-protective value.

Many jobs are going to present workers with daily brain challenges. These may include having data to enter, information to process, items to remember, things to multi-task, questions to answers, and so on.[23]

Now, if your nine-to-five has you on the verge of a panic-induced coronary, then you might want to seek stimulation elsewhere. But if your day job isn’t overly stressful or soul-sucking, then realize that it might be giving your brain an assist.

Caveats

When it comes to Alzheimer’s prevention, there are three divergent perspectives on the efficacy of mental exercise. These are as follows. (1) Mental exercise is possibly helpful. (2) Mental exercise is likely neither helpful not harmful. (3) Mental exercise is potentially harmful.

Objections

The third position – that mental can be potentially harmful – suggests a few objections to the strategies outlined above.

False Hope?

Firstly, some investigators worry that these considerations might give a person “false hope.” The idea, here, is – presumably – that someone might form beliefs such as that doing crossword puzzles has the power to confer some sort of magical protection against dementia, or that doing them could even reverse the disease. Sadly, these don’t seem to be the case.

But it seems to me that the solution is to have realistic expectations, rather than abandoning the idea of doing mental exercises.

Ineffective?

Secondly, and relatedly, some people object that these interventions are just plain ineffective. For example, in some studies – like regarding bilingualism – participants ended up getting Alzheimer’s anyway.

But this shouldn’t mean that the interventions are without value. It may be that we have to clarify what we mean by “effective.” If “effective” has to mean 100% protection against Alzheimer’s, then we might have to confess these interventions to be “ineffective.” But could mental exercises be “effective” at delaying Alzheimer’s?

Delaying onset of a disease seems valuable in and of itself. For example, if you can maintain a higher quality of life longer, wouldn’t you want to do it?

So, maybe playing checkers or working won’t guarantee that I never get Alzheimer’s. But if they (and other things) can help me to push onset back 2 years, 4 years, 6 years… it’s worth it to me.[24]

However, some people mention another facet of this objection. To put it directly, it’s possible that “incipient” or as-of-yet undetected dementia might prompt people to withdraw from social situations and to cease engaging in mentally stimulating activities.

On this picture, it’s not so much that you should exercise your brain to ward off Alzheimer’s. It’s more that once you reduce your level of mental engagement, it’s likely that you have Alzheimer’s – latently – already.

Of course, it is true that I don’t have any special insight into the mechanics or direction of the causation – if any – between mental exercise and dementia. It could be that dementia causes a lack of mental exercise; it could be that a lack of brain engagement causes dementia; it could be that they both have a third, presently unknown cause; or it could be that they are causally unrelated.

Still… only one of those possibilities suggests any direct way for me to influence my mental health positively. In the absence of some impelling reason for me to think that brain exercise isn’t at least possibly beneficial to me, I’ll continue to operate as though it might.

Counterproductive?

Thirdly, some commentators have spoken (or written) in such a way as to suggest that brain exercises could actually be harmful! A few titles make statements such as that mental training can “speed up dementia.” A few acknowledge that mental stimulation might buy time, but that it also accelerates decline once it begins.

There are a few things to be said.

Number one, insofar as these statements make it seem as if someone could be worse off for having exercised their brains, these summaries are a bit misleading. The “acceleration” of the decline can be explained as a simple matter of mathematics, provided only that the dementia is at least partially a matter of biology or physiology.

What I mean is this. Mental exercises almost certainly help boost or preserve cognitive function. But Alzheimer’s involves literal, physical damage to the brain. So, ultimately, mental exercises cannot undo physical damage.

However, through things such as by increasing “cognitive reserve,” they may be able to stave off the noticeable effects of the condition. But this means that once the effects of the condition do become noticeable, the disease may be “compressed,” and the decline may appear to be more rapid or steeper than it would have been otherwise.

Mathematically, this means that the decline is “quicker” either in that it happens over a shorter time, or that it occurs from a higher “starting point” – or both. This can be seen fairly readily from a simple curved-line graph.

In the graph, I show four different trajectories, all ending at age 80.

Red line: no exercise

The red line represents a person who doesn’t exercise at all, and whose decline begins at age 70. The decline concludes at age 80 – as it will for each of the four imagined scenarios.

Blue line: exercise preserves brain function

The blue line represents a person whose exercise preserves their cognitive function an extra five years. So, their decline begins at age 75. It still concludes at age 80.

Orange line: exercise increases, but doesn’t preserve

The orange line represents a person for whom exercise gives their brain function a boost. I didn’t also assume that this boost bought them any additional time. So, you see their decline begins at the same point as the person who doesn’t exercise at all: age 70. This is the person who has a “higher starting point.” The decline also ends at age 80.

Green line: exercise increased brain function

Finally, the green line represents the person for whom exercise both gives a boost to brain function and preserves it. Obviously, this is the best-case scenario. Since the brain function is boosted, the starting point is higher. Since it is preserved, I have their decline begin at age 75. Like everyone else, it stops at age 80.

Analysis

In this toy model, I have envisioned four scenarios, representing four possible combinations. (1) No boost to brain function and no preservation of brain function;[25] (2) preservation of brain function with no boost; (3) no preservation of function, but some boost; and (4) both preservation and boost.

In each of the four cases, we’re looking at people between the ages of 65 and 80. I have assigned arbitrary “brain-function points” between 100 and 400.[26] Furthermore, I have supposed that people start to decline beginning at age 70 or 75, depending on whether there is preservation or not.[27]

(You could either see these as representing four different, but relevantly similar, people. Or you could see it as representing four different possible trajectories for one and the same person. I prefer the latter.)

The four resulting combinations are as follows.

No boost, no preservation

The red line depicts a person who doesn’t engage in any mental exercise at all. The decline begins at level “300” (just an arbitrary number) and ends at level “100.” This is a difference of 200 points. It takes ten years, which means that they lose twenty points a year.

No boost, preservation

The blue line buys the person an extra five years of preservation. Since they hit the same level – level “100” – at the end, their decline occurs twice as fast as for the person who didn’t exercise. They drop 40 points per year, which is twice the rate of decline. This is because the same amount of decline (as occurred with red) is compressed into half the time.

Boost, no preservation

The orange line shows a person with a bit of a boost (getting them to 400), but no extra time before decline begins. They start higher, but end in the same place, dropping 300 points in ten years. This yields a rate of 30 points per year. The amount of decline (compared with red) is 1.5 times greater (150%) but is stretched over the same length of time (as red).

Boost, preservation

The green line shows a person with both boost and preservation. This person bought an extra five years before visible decline. But they also have the extra “100 points” of function. So, their decline starts at a later age (compared to red) – age 75 – and from a higher starting point (again, compared with red) – 400 points. Since they decline 300 points over five years, their rate of decline is 60 points per year.

Conclusion

That we see “higher rates” of decline in the exercisers is due to either (or both) of two factors.

Factor 1: The decline happens over a shorter span of time (as with blue and green); or…

Factor 2: The decline happens from a higher starting point (as with orange and green).

I said earlier that the explanation for the higher decline rates was mathematical. When a predetermined amount of decline happens over a shorter time frame, the rate of decline is increased. This is mathematical in this sense. Take some number, n. n divided by 5 is going to be bigger than n divided by 10.

Moreover, when a predetermined endpoint of decline is reached from a higher beginning point, the slope of the line representing that decline is steeper.[28] This is also mathematical, since the slope of a line is merely a value (m) in the equation representing that line. So, if the cognitive “drop off” is steeper, all we’re saying is that the value of slope (m) for that drop off is a bigger number than it is if the drop off were not as steep.

At the end of the day, for me, I would rather have my cognitive function preserved for as long as possible – and boosted as high as possible – even if I experience an eventual decline.[29]

Curiously, you could even argue that having a “quicker” or “higher” rate of decline is preferable to a slower rate in that it likely saves caretaker energy as well as money devoted to care!

Training Is Parochial

Fourthly, you may read that certain forms of “brain training” are very limited in terms of what they accomplish. Even where certain mental exercises may be worthwhile, their impact may be restricted. To put it another way, specific benefits may not generalize to other areas of your daily or mental life.

For example, reading books may help boost your processing speed, but maybe doesn’t help enhance your memory. (It’s just an illustration; I don’t know whether it does or doesn’t.)

Somewhere I read a researcher giving the following analogy. Some brain exercises can be likened to working out physically by doing only one or two exercises. These exercises – like bicep curls – may strengthen a single muscle (the biceps), but they are unlikely to impact the overall health of the body much.

A few things may be said in reply. Number one, you can make the case that doing a few exercises is better than doing none. A person who does biceps curls may not be as fit or healthy as a person who trains his or her whole body. But he or she may well be more fit or healthy than he or she would be if they did nothing at all.

Number two, whether a given exercise has broad or narrow impact may depend on the sort of exercise being done. In physical training, there are differences between compound and isolation exercises. It’s one thing to do bicep curls or grip strengtheners all day long. It’s another to do deadlifts or squats. The former may only affect one or two muscles; the latter might well affect the entire body. It is doubtful that we know enough about “brain training” to really understand the broader impact of a lot of the mental exercises discussed here. For example, is playing chess more than doing bicep curls, or more like doing squats? I’m not sure. And I’m not sure that anyone else is sure, either!

Blame the Victim?

Yet another objection, fifthly, is that talking about mental exercise may lead to sufferers being “blamed” for their Alzheimer’s. The idea here is that some people might conclude that if John Doe has dementia, then he must have been mentally inactive or lazy.

Sometimes you may read comparisons to smoking. People who smoke are at higher risk of lung cancer. So, if a smoker gets lung cancer, then he or she assumes some of the responsibility for that condition.

By way of response, I should first remind readers that Alzheimer’s risk almost certainly has a – probably a significant – genetic component. (See my video about risk factors HERE; or read the article on the same topic HERE.) To put it differently, some people are simply more at risk than others of developing it.

Having said that, I will simply repeat what I have mentioned many times in my written and video-graphic work: I am trying to stack the odds in my favor. I realize that if I smoke, I’ll be at increased risk for lung cancer. Although the data may not be as clear cut for the relationship between mental exercise and dementia, I’ll say that for me personally I’d rather exercise, and have it avail me nothing, than not exercise and have it turn out that it would have helped me.

If other people value other things over exercising, then I would suggest that it is their prerogative to do so. In the first place, the data in favor of mental exercise is not so compelling as to make it undeniable that it helps preserve or boost cognitive function or that it can ward off Alzheimer’s.

But even if the data were that compelling, it’s not clear that someone has to value preserving or boosting cognitive function or must value warding off Alzheimer’s, over not doing any of these. I confess that such a position would be foreign to my own thinking. But it’s not something that moves me to start throwing words like “blame” around.

I suppose you could put my answer this way. If a person doesn’t perform mental exercises, it’s either because they don’t think it will help or they don’t care if it helps or not. If they don’t think it will help, then their choice not to exercise is rational. They have discharged their rational duty and it’s not obvious to me that there’s anything to blame them for.

If they don’t care, then the choice itself may be irrational (i.e., not rational). But it’s not clear why a person choosing irrationally in this way wouldn’t care if exercising helps but would care if they’re “blamed” for not caring. It seems to me more likely (or at least more consistent) if they didn’t care about either one. So, even if the choice is blameworthy, it doesn’t appear to have the result the objector is worried about. It seems that the concern in the objection is centered on the perceived hurt feelings of the person being blamed. But, to reiterate: for all we know, the person who doesn’t care about not exercising wouldn’t care about being blamed for not exercising. If this is so, then it’s not obvious that there would be any hurt feelings for us to worry about.

Conclusions (Tentative)

One article ventured the opinion “that lifestyle choices may even counteract genetic predisposition for Alzheimer’s.” If true, that’s huge.[30] And it would put a lot of control in our hands.

Here are a few takeaways.

Train the Whole Brain

But staying mentally fit and sharp may really come down to neural recruitment: using multiple parts of your brain, not just a few.

Be Consistent

It’s also going to involve consistency. Many reports mention the need to engage in stimulating activities regularly – say two or more times weekly – not just every blue moon.

Try Something New

Another key element is novelty. Sometimes trying something new may be more valuable than doing the same things over and over. There may be two “levels” of novelty. Think about some of the things on this list. For example, chess or reading. Every game of chess you play has the possibility of being different from every other game. And if you read new articles or books every day, you are adding some variety. However, we might call this low-level variety. A higher level of variety can be attained if you learn a new language or musical instrument, for example. Interestingly, there may be a kind of middle level as well. For example, a person could switch from reading fiction to nonfiction, or from reading prose to reading poetry.

Act as Though It’s ‘Use It Or Lose It’

As the Independent put it: “use it or lose it” idea may just “give a person a ‘higher starting point’ from which to decline.” But this still seems advantageous.

Realize: ‘Better Late Than Never’

Some commentators express the message that its always “better late than never.” But you should probably take the position that it’s desirable to start now! This applies to you whether you are a sufferer or a person looking to avoid the condition altogether.

No Silver Bullets

Still, neither I nor most other researchers are suggesting that any of these measures amounts to a “cure.”

Aim to Have a Healthy Lifestyle

Additionally, these mental activities almost certainly need to be situated in a larger context – a “lifestyle package,” as it were. Genetic predisposition notwithstanding, if you really want to stack the odds in your favor, you’ll need to address your blood pressure, body mass, cholesterol, diet, level of physical exercise, and sleep patterns.

I can tell you that I’m implementing a number of these measures today. Most of the items on this list are cheap (or free) and easy to obtain. And after all that’s been said, I think it’s reasonable to maintain that they can’t hurt. And some of them just might help. So…go on: give your brain a good workout!


[1] See, e.g., Chiara Giordano, “Doing Sudoku and Crosswords Won’t Stop Dementia or Mental Decline, Study Suggests,” Dec. 11, 2018, <https://www.independent.co.uk/news/health/sudoku-crosswords-dementia-mental-decline-brain-study-aberdeen-university-research-a8677466.html>.

[2] Giordano, loc. cit.

[3] Felix Gussone, “5 Things You Didn’t Know About Alzheimer’s,” CNN, Jul. 17, 2014, <https://www.cnn.com/2014/07/14/health/alzheimers-disease-conference/index.html>.

[4] See, e.g., Margaret Gatz, Educating the Brain to Avoid Dementia: Can Mental Exercise Prevent Alzheimer Disease?” Public Library of Science, vol. 2, no. 1, Jan. 25, 2005, p. e7, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC545200/>.

[5] Amy Marcus, “How to Outsmart Alzheimer’s,” Wall Street Journal, Mar. 30, 2010, <https://www.wsj.com/articles/SB10001424052748703416204575145921517534304>.

[6] Allison Aubrey, “Mental Stimulation Postpones, Then Speeds Dementia,” National Public Radio, Weekend Ed. Saturday, Sept. 4, 2010, <https://www.npr.org/templates/story/story.php?storyId=129628082>.

[7] Ivan Vega, “‘Checkmate the Onset of Dementia’: Prescribing Chess to Elderly People as a Primary Prevention of Dementia,” Journal of Alzheimer’s Disease, Jan. 25, 2019, <https://www.j-alz.com/editors-blog/posts/checkmate-onset-dementia>.

[8] Gussone, loc. cit.

[9] According to Jagan Pillai, Charles Hall, Dennis Dickson, Herman Buschke, Richard Lipton, and Joe Verghese, “Association of Crossword Puzzle Participation with Memory Decline in Persons Who Develop Dementia,” Journal of the International Neuropsychological Society, vol. 17, no. 6, Nov., 2011, <https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3885259/>.

[10] At least, this is the assertion of the weblog Puerto Rico Report, in the post “Bilingual America,” Aug. 11, 2017, <https://www.puertoricoreport.com/bilingual-america>.

[11] Ingrid Piller, “Multilingual Europe,” Language on the Move, Jul. 18, 2012, <https://www.languageonthemove.com/multilingual-europe/>.

[12] The precise time of onset can be extremely difficult to identify.

[13] Ryan Jaslow, “Learning Another language May Help Delay Dementia,” CBS, Nov. 6, 2013, <https://www.cbsnews.com/news/learning-another-language-may-help-delay-dementia/>.

[14] Ibid.

[15] Giordano, loc. cit.

[16] Rob Nelson, “Hidden Heroes: Queens 12-Year-Old Helping People With Alzheimer’s,” ABC News, Apr. 26, 2019, <https://abc7ny.com/health/hidden-heroes-queens-12-year-old-helps-people-with-alzheimers/5272644/>.

[17] Though, for the counterpoint that online reading may be detrimental, see “‘The Shallows’: This Is Your Brain Online,” National Public Radio, All Things Considered, Jun. 2, 2010, <https://www.npr.org/templates/story/story.php?storyId=127370598>.

[18] “A Brain Scientist Who Studies Alzheimer’s Explains How She Stays Mentally Fit,” National Public Radio, Morning Ed., Oct. 8, 2018, <https://www.npr.org/sections/health-shots/2018/10/08/654903558/a-brain-scientist-who-studies-alzheimers-explains-how-she-stays-mentally-fit>.

[19] “A Brain Scientist Who Studies Alzheimer’s Explains How She Stays Mentally Fit,” loc. cit.

[20] “Sudoku or Crosswords May Help Keep Your Brain 10 Years Younger,” Healthline, n.d., <https://www.healthline.com/health-news/can-sudoku-actually-keep-your-mind-sharp>.

[21] Helen Brooker, Keith Wesnes, Clive Ballard, Adam Hampshire, Dag Aarsland, Zunera Khan, Rob Stenton, Maria Megalogeni, and Anne Corbett, “The Relationship Between the Frequency of Number‐Puzzle Use and Baseline Cognitive Function in a Large Online Sample of Adults Aged 50 and Over,” International Journal of Geriatric Psychiatry, vol. 34, no. 7, publ. in print Jul. 2019, pp. 932-940, publ. online Feb. 11, 2019, <https://onlinelibrary.wiley.com/doi/abs/10.1002/gps.5085>.

[22] “A Brain Scientist Who Studies Alzheimer’s Explains How She Stays Mentally Fit,” loc. cit.

[23] Ibid.

[24] Again, onset detection is not a little tricky.

[25] Both ideas – “boosting brain function” and “preserving brain function” – are a little vague and would need to be sharpened to be of greater use. However, my model is merely trying to show that the “higher rates of decline” spoken about in some articles might well be nothing to worry about. So, I have abstracted away from some of the details because I don’t think they’re necessary for the point.

[26] This raises the issue of how we would actually be able to measure cognitive ability. There are various assessment tests. But it is possible that these assessments fail, for one reason or other, to paint a true or complete picture of a person’s cognitive situation. This is simply a model.

[27] This choice was arbitrary.

[28] I realize that I opted to display the graph with curved lines. This was simply an esthetic choice since when I used straight lines, the lines overlapped in places and couldn’t be easily distinguished. The information is simply sample and hypothetical data for illustrative purposes only. It could be represented with straight lines. And if it were represented this way, then the resulting lines would have calculable slopes in the usual sense.

[29] As a side note, the red line also represents a case in which a person exercises, but it fails to boost their brain function or preserve it at all. So, you’ll notice that if the exercises are utterly ineffective, you’re no worse off than you would be had you not exercised at all. You might think that you would have wasted your time. I suppose this boils down to whether you find any of the exercises enjoyable – or potentially enjoyable – or not. But even still, personally, it strikes me as improbable that mental exercises would do nothing whatsoever. Readers may think differently.

[30] More scientific study and philosophical reflection is needed, however. Some studies abstract away from possibly relevant data, including economic, educational, genetic, intelligence, and sociological factors.